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Rafat A. Siddiqui

Cellular Biochemistry Laboratory

Methodist Research Institute

Indiana University Health

Indianapolis

USA

[email]@clarian.org

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Cellular Biochemistry Laboratory, Methodist Research Institute, Indiana University Health, Indianapolis, USA. 2011
  • Methodist Research Institute, Clarian Health Partners, Indianapolis, IN 46202, USA. 2001 - 2009

References

  1. A synergistic antiproliferation effect of curcumin and docosahexaenoic acid in SK-BR-3 breast cancer cells: unique signaling not explained by the effects of either compound alone. Altenburg, J.D., Bieberich, A.A., Terry, C., Harvey, K.A., Vanhorn, J.F., Xu, Z., Jo Davisson, V., Siddiqui, R.A. BMC. Cancer (2011) [Pubmed]
  2. Attenuation of proteolysis and muscle wasting by curcumin c3 complex in MAC16 colon tumour-bearing mice. Siddiqui, R.A., Hassan, S., Harvey, K.A., Rasool, T., Das, T., Mukerji, P., DeMichele, S. Br. J. Nutr. (2009) [Pubmed]
  3. n-3 fatty acids prevent whereas trans-fatty acids induce vascular inflammation and sudden cardiac death. Siddiqui, R.A., Harvey, K.A., Ruzmetov, N., Miller, S.J., Zaloga, G.P. Br. J. Nutr. (2009) [Pubmed]
  4. Modulation of enzymatic activities by n-3 polyunsaturated fatty acids to support cardiovascular health. Siddiqui, R.A., Harvey, K.A., Zaloga, G.P. J. Nutr. Biochem. (2008) [Pubmed]
  5. Anticancer properties of oxidation products of docosahexaenoic acid. Siddiqui, R.A., Harvey, K., Stillwell, W. Chem. Phys. Lipids (2008) [Pubmed]
  6. Modulation of lipid rafts by Omega-3 fatty acids in inflammation and cancer: implications for use of lipids during nutrition support. Siddiqui, R.A., Harvey, K.A., Zaloga, G.P., Stillwell, W. Nutr. Clin. Pract (2007) [Pubmed]
  7. Anticancer properties of propofol-docosahexaenoate and propofol-eicosapentaenoate on breast cancer cells. Siddiqui, R.A., Zerouga, M., Wu, M., Castillo, A., Harvey, K., Zaloga, G.P., Stillwell, W. Breast Cancer Res. (2005) [Pubmed]
  8. Inhibition of phenylephrine-induced cardiac hypertrophy by docosahexaenoic acid. Siddiqui, R.A., Shaikh, S.R., Kovacs, R., Stillwell, W., Zaloga, G. J. Cell. Biochem. (2004) [Pubmed]
  9. Omega 3-fatty acids: health benefits and cellular mechanisms of action. Siddiqui, R.A., Shaikh, S.R., Sech, L.A., Yount, H.R., Stillwell, W., Zaloga, G.P. Mini. Rev. Med. Chem (2004) [Pubmed]
  10. Cell-cycle arrest in Jurkat leukaemic cells: a possible role for docosahexaenoic acid. Siddiqui, R.A., Jenski, L.J., Harvey, K.A., Wiesehan, J.D., Stillwell, W., Zaloga, G.P. Biochem. J. (2003) [Pubmed]
  11. Prevention of docosahexaenoic acid-induced cytotoxicity by phosphatidic acid in Jurkat leukemic cells: the role of protein phosphatase-1. Siddiqui, R.A., Jenski, L.J., Wiesehan, J.D., Hunter, M.V., Kovacs, R.J., Stillwell, W. Biochim. Biophys. Acta (2001) [Pubmed]
 
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