Klaus Ruckdeschel
Max von Pettenkofer Institute for Hygiene and Medical Microbiology
80336 Munich
Germany
[email]@*.mpk.med.uni-muenchen.de
Name/email consistency: high
- Signaling of apoptosis through TLRs critically involves toll/IL-1 receptor domain-containing adapter inducing IFN-beta, but not MyD88, in bacteria-infected murine macrophages. Ruckdeschel, K., Pfaffinger, G., Haase, R., Sing, A., Weighardt, H., Häcker, G., Holzmann, B., Heesemann, J. J. Immunol. (2004)
- Divergence of apoptosis-inducing and preventing signals in bacteria-faced macrophages through myeloid differentiation factor 88 and IL-1 receptor-associated kinase members. Ruckdeschel, K., Mannel, O., Schröttner, P. J. Immunol. (2002)
- Immunomodulation of macrophages by pathogenic Yersinia species. Ruckdeschel, K. Arch. Immunol. Ther. Exp. (Warsz.) (2002)
- Lipopolysaccharide desensitization of macrophages provides protection against Yersinia enterocolitica-induced apoptosis. Ruckdeschel, K., Richter, K. Infect. Immun. (2002)
- Arginine-143 of Yersinia enterocolitica YopP crucially determines isotype-related NF-kappaB suppression and apoptosis induction in macrophages. Ruckdeschel, K., Richter, K., Mannel, O., Heesemann, J. Infect. Immun. (2001)