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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)

Ruth Halaban

Yale University School of Medicine

Department of Dermatology


P.O. Box: 208059



Name/email consistency: high



  • Yale University School of Medicine, Department of Dermatology, HRT604A, P.O. Box: 208059, USAe-mail:. 2010
  • Department of Dermatology, Yale University School of Medicine, New Haven, Connecticut, United States of America. 1997 - 2009


  1. Meenhard herlyn. Halaban, R. Pigment. Cell. Melanoma. Res (2010) [Pubmed]
  2. Integrative analysis of epigenetic modulation in melanoma cell response to decitabine: clinical implications. Halaban, R., Krauthammer, M., Pelizzola, M., Cheng, E., Kovacs, D., Sznol, M., Ariyan, S., Narayan, D., Bacchiocchi, A., Molinaro, A., Kluger, Y., Deng, M., Tran, N., Zhang, W., Picardo, M., Enghild, J.J. PLoS. ONE (2009) [Pubmed]
  3. Rb/E2F: a two-edged sword in the melanocytic system. Halaban, R. Cancer Metastasis Rev. (2005) [Pubmed]
  4. Abnormal acidification of melanoma cells induces tyrosinase retention in the early secretory pathway. Halaban, R., Patton, R.S., Cheng, E., Svedine, S., Trombetta, E.S., Wahl, M.L., Ariyan, S., Hebert, D.N. J. Biol. Chem. (2002) [Pubmed]
  5. Coexpression of wild-type tyrosinase enhances maturation of temperature-sensitive tyrosinase mutants. Halaban, R., Cheng, E., Hebert, D.N. J. Invest. Dermatol. (2002) [Pubmed]
  6. Pigmentation in melanomas: changes manifesting underlying oncogenic and metabolic activities. Halaban, R. Oncol. Res. (2002) [Pubmed]
  7. Proper folding and endoplasmic reticulum to golgi transport of tyrosinase are induced by its substrates, DOPA and tyrosine. Halaban, R., Cheng, E., Svedine, S., Aron, R., Hebert, D.N. J. Biol. Chem. (2001) [Pubmed]
  8. Deregulated E2F transcriptional activity in autonomously growing melanoma cells. Halaban, R., Cheng, E., Smicun, Y., Germino, J. J. Exp. Med. (2000) [Pubmed]
  9. The regulation of normal melanocyte proliferation. Halaban, R. Pigment Cell Res. (2000) [Pubmed]
  10. Endoplasmic reticulum retention is a common defect associated with tyrosinase-negative albinism. Halaban, R., Svedine, S., Cheng, E., Smicun, Y., Aron, R., Hebert, D.N. Proc. Natl. Acad. Sci. U.S.A. (2000) [Pubmed]
  11. Melanoma cell autonomous growth: the Rb/E2F pathway. Halaban, R. Cancer Metastasis Rev. (1999) [Pubmed]
  12. Aberrant retention of tyrosinase in the endoplasmic reticulum mediates accelerated degradation of the enzyme and contributes to the dedifferentiated phenotype of amelanotic melanoma cells. Halaban, R., Cheng, E., Zhang, Y., Moellmann, G., Hanlon, D., Michalak, M., Setaluri, V., Hebert, D.N. Proc. Natl. Acad. Sci. U.S.A. (1997) [Pubmed]
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