Roy E. Weiss
Section of Adult and Pediatric Endocrinology
Diabetes
and Metabolism
Department of Medicine
USA
Name/email consistency: high
- Approach to the patient with resistance to thyroid hormone and pregnancy. Weiss, R.E., Dumitrescu, A., Refetoff, S. J. Clin. Endocrinol. Metab. (2010)
- Thyroid function and effect of aging in combined hetero/homozygous mice deficient in thyroid hormone receptors alpha and beta genes. Weiss, R.E., Chassande, O., Koo, E.K., Macchia, P.E., Cua, K., Samarut, J., Refetoff, S., Refetoff, S. J. Endocrinol. (2002)
- Thyroid nodules: diagnosis and therapy. Weiss, R.E., Lado-Abeal, J. Curr. Opin. Oncol (2002)
- Thyroid function in mice with compound heterozygous and homozygous disruptions of SRC-1 and TIF-2 coactivators: evidence for haploinsufficiency. Weiss, R.E., Gehin, M., Xu, J., Sadow, P.M., O'Malley, B.W., Chambon, P., Refetoff, S. Endocrinology (2002)
- Thyroid hormone and cardiac function in mice deficient in thyroid hormone receptor-alpha or -beta: an echocardiograph study. Weiss, R.E., Korcarz, C., Chassande, O., Cua, K., Sadow, P.M., Koo, E., Samarut, J., Lang, R. Am. J. Physiol. Endocrinol. Metab. (2002)
- A serpentine thyroid. Weiss, R.E. Thyroid (2002)
- Clear fluid from a thyroid cyst. Weiss, R.E. Thyroid (2000)
- Resistance to thyroid hormone. Weiss, R.E., Refetoff, S. Rev. Endocr. Metab. Disord (2000)
- Mice deficient in the steroid receptor co-activator 1 (SRC-1) are resistant to thyroid hormone. Weiss, R.E., Xu, J., Ning, G., Pohlenz, J., O'Malley, B.W., Refetoff, S. EMBO J. (1999)
- Thyroid hormone action on liver, heart, and energy expenditure in thyroid hormone receptor beta-deficient mice. Weiss, R.E., Murata, Y., Cua, K., Hayashi, Y., Seo, H., Refetoff, S. Endocrinology (1998)
- Thyrotropin regulation by thyroid hormone in thyroid hormone receptor beta-deficient mice. Weiss, R.E., Forrest, D., Pohlenz, J., Cua, K., Curran, T., Refetoff, S. Endocrinology (1997)
- Dominant inheritance of resistance to thyroid hormone not linked to defects in the thyroid hormone receptor alpha or beta genes may be due to a defective cofactor. Weiss, R.E., Hayashi, Y., Nagaya, T., Petty, K.J., Murata, Y., Tunca, H., Seo, H., Refetoff, S. J. Clin. Endocrinol. Metab. (1996)