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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

Saadia Bashir Hassan

Division of Clinical Pharmacology

Department of Medical Sciences

University Hospital

751 85 Uppsala

Sweden

[email]@medsci.uu.se

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Division of Clinical Pharmacology, Department of Medical Sciences, University Hospital, 751 85 Uppsala, Sweden. 2005 - 2010
  • Division of Clinical Pharmacology, Uppsala University, Sweden. 2001

References

  1. Alpha terpineol: a potential anticancer agent which acts through suppressing NF-kappaB signalling. Hassan, S.B., Gali-Muhtasib, H., Göransson, H., Larsson, R. Anticancer Res. (2010) [Pubmed]
  2. Primary lymphocytes as predictors for species differences in cytotoxic drug sensitivity. Hassan, S.B., Haglund, C., Aleskog, A., Larsson, R., Lindhagen, E. Toxicol. In. Vitro (2007) [Pubmed]
  3. CHS 828 kill tumour cells by inhibiting the nuclear factor-kappaB translocation but unlikely through down-regulation of proteasome. Hassan, S.B., Lövborg, H., Lindhagen, E., Karlsson, M.O., Larsson, R. Anticancer Res. (2006) [Pubmed]
  4. Cytotoxic activity of a new paclitaxel formulation, Pacliex, in vitro and in vivo. Hassan, S., Dhar, S., Sandström, M., Arsenau, D., Budnikova, M., Lokot, I., Lobanov, N., Karlsson, M.O., Larsson, R., Lindhagen, E. Cancer Chemother. Pharmacol. (2005) [Pubmed]
  5. A hollow fiber model for in vitro studies of cytotoxic compounds: activity of the cyanoguanidine CHS 828. Hassan, S.B., de la Torre, M., Nygren, P., Karlsson, M.O., Larsson, R., Jonsson, E. Anticancer. Drugs (2001) [Pubmed]
  6. Model for time dependency of cytotoxic effect of CHS 828 in vitro suggests two different mechanisms of action. Hassan, S.B., Jonsson, E., Larsson, R., Karlsson, M.O. J. Pharmacol. Exp. Ther. (2001) [Pubmed]
 
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