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Junichi Sadoshima

Cardiovascular Research Institute

University of Medicine and Dentistry of New Jersey

New Jersey Medical School

185 South Orange Ave

[email]@umdnj.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Cardiovascular Research Institute, University of Medicine and Dentistry of New Jersey, New Jersey Medical School, 185 South Orange Ave. 2002 - 2012
  • Department of Cell Biology and Molecular Medicine, UMDNJ-New Jersey Medical School, 185 South Orange Avenue, Newark. 2011

References

  1. Rheb is a Critical Regulator of Autophagy During Myocardial Ischemia: Pathophysiological Implications in Obesity and Metabolic Syndrome. Sciarretta, S., Zhai, P., Shao, D., Maejima, Y., Robbins, J., Volpe, M., Condorelli, G., Sadoshima, J. Circulation (2012) [Pubmed]
  2. Endogenous Muscle Atrophy F-Box Mediates Pressure Overload-Induced Cardiac Hypertrophy Through Regulation of Nuclear Factor-{kappa}B. Usui, S., Maejima, Y., Pain, J., Hong, C., Cho, J., Park, J.Y., Zablocki, D., Tian, B., Glass, D.J., Sadoshima, J. Circ. Res. (2011) [Pubmed]
  3. Differential Roles of GSK-3{beta} During Myocardial Ischemia and Ischemia/Reperfusion. Zhai, P., Sciarretta, S., Galeotti, J., Volpe, M., Sadoshima, J. Circ. Res. (2011) [Pubmed]
  4. The role of autophagy during ischemia/reperfusion. Sadoshima, J. Autophagy (2008) [Pubmed]
  5. Inhibition of endogenous thioredoxin in the heart increases oxidative stress and cardiac hypertrophy. Yamamoto, M., Yang, G., Hong, C., Liu, J., Holle, E., Yu, X., Wagner, T., Vatner, S.F., Sadoshima, J. J. Clin. Invest. (2003) [Pubmed]
  6. The MEKK1-JNK pathway plays a protective role in pressure overload but does not mediate cardiac hypertrophy. Sadoshima, J., Montagne, O., Wang, Q., Yang, G., Warden, J., Liu, J., Takagi, G., Karoor, V., Hong, C., Johnson, G.L., Vatner, D.E., Vatner, S.F. J. Clin. Invest. (2002) [Pubmed]
 
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