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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

J.I. Satoh

Division of Neurology

Department of Internal Medicine

Saga Medical School

Japan

[email]@*.saga-med.ac.jp

Name/email consistency: high

 
 
 
 
 
 
 

Affiliation

  • Division of Neurology, Department of Internal Medicine, Saga Medical School, Japan. 2000 - 2002

References

  1. A putative polymorphic Val44Ala variation in the synphilin-1 gene is undetectable in Japanese sporadic Parkinson's disease patients. Satoh, J.I., Kuroda, Y. Eur. J. Neurol. (2002) [Pubmed]
  2. Cytokines and neurotrophic factors fail to affect Nogo-A mRNA expression in differentiated human neurones: implications for inflammation-related axonal regeneration in the central nervous system. Satoh, J.I., Kuroda, Y. Neuropathol. Appl. Neurobiol. (2002) [Pubmed]
  3. Nicastrin, a key regulator of presenilin function, is expressed constitutively in human neural cell lines. Satoh, J., Kuroda, Y. Neuropathology (2001) [Pubmed]
  4. Differing effects of IFN beta vs IFN gamma in MS: gene expression in cultured astrocytes. Satoh, J., Kuroda, Y. Neurology (2001) [Pubmed]
  5. A polymorphic variation of serine to tyrosine at codon 18 in the ubiquitin C-terminal hydrolase-L1 gene is associated with a reduced risk of sporadic Parkinson's disease in a Japanese population. Satoh, J., Kuroda, Y. J. Neurol. Sci. (2001) [Pubmed]
  6. Gene expression profile in prion protein-deficient fibroblasts in culture. Satoh, J., Kuroda, Y., Katamine, S. Am. J. Pathol. (2000) [Pubmed]
  7. Beta-catenin expression in human neural cell lines following exposure to cytokines and growth factors. Satoh, J., Kuroda, Y. Neuropathology (2000) [Pubmed]
  8. Amyloid precursor protein beta-secretase (BACE) mRNA expression in human neural cell lines following induction of neuronal differentiation and exposure to cytokines and growth factors. Satoh, J., Kuroda, Y. Neuropathology (2000) [Pubmed]
 
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