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Jun-ichi Satoh

Department of Immunology

National Institute of Neuroscience

NCNP, Kodaira

Tokyo

Japan

[email]@ncnp.go.jp

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Affiliations

Department of Immunology, National Institute of Neuroscience, NCNP, Kodaira, Tokyo, Japan. 2002 - 2006
Department of Bioinformatics, Meiji Pharmaceutical University, 2-522-1 Noshio, Kiyose, Japan. 2006

References

Rapid identification of 14-3-3-binding proteins by protein microarray analysis. Satoh, J., Nanri, Y., Yamamura, T. J. Neurosci. Methods (2006) [Pubmed]
Microarray analysis identifies a set of CXCR3 and CCR2 ligand chemokines as early IFNbeta-responsive genes in peripheral blood lymphocytes in vitro: an implication for IFNbeta-related adverse effects in multiple sclerosis. Satoh, J., Nanri, Y., Tabunoki, H., Yamamura, T. BMC. Neurol (2006) [Pubmed]
Nogo-A and nogo receptor expression in demyelinating lesions of multiple sclerosis. Satoh, J., Onoue, H., Arima, K., Yamamura, T. J. Neuropathol. Exp. Neurol. (2005) [Pubmed]
The 14-3-3 protein forms a molecular complex with heat shock protein Hsp60 and cellular prion protein. Satoh, J., Onoue, H., Arima, K., Yamamura, T. J. Neuropathol. Exp. Neurol. (2005) [Pubmed]
The 14-3-3 protein epsilon isoform expressed in reactive astrocytes in demyelinating lesions of multiple sclerosis binds to vimentin and glial fibrillary acidic protein in cultured human astrocytes. Satoh, J., Yamamura, T., Arima, K. Am. J. Pathol. (2004) [Pubmed]
Gene expression profile following stable expression of the cellular prion protein. Satoh, J., Yamamura, T. Cell. Mol. Neurobiol. (2004) [Pubmed]
Detection of the 14-3-3 protein in the cerebrospinal fluid of Japanese multiple sclerosis patients presenting with severe myelitis. Satoh, J., Yukitake, M., Kurohara, K., Takashima, H., Kuroda, Y. J. Neurol. Sci. (2003) [Pubmed]
The constitutive and inducible expression of Nurr1, a key regulator of dopaminergic neuronal differentiation, in human neural and non-neural cell lines. Satoh, J., Kuroda, Y. Neuropathology (2002) [Pubmed]