Daniel R. Scoles
Women's Cancer Research Institute
CSMC Burns and Allen Research Institute
Cedars-Sinai Medical Center
8700 Beverly Boulevard
USA
Name/email consistency: high
- The merlin interacting proteins reveal multiple targets for NF2 therapy. Scoles, D.R. Biochim. Biophys. Acta (2008)
- Characterization of CSOC 882, a novel immortalized ovarian cancer cell line expressing EGFR, HER2, and activated AKT. Scoles, D.R., Pavelka, J., Cass, I., Tran, H., Baldwin, R.L., Armstrong, K., Karlan, B.Y. Gynecol. Oncol. (2007)
- Schwannomin inhibits tumorigenesis through direct interaction with the eukaryotic initiation factor subunit c (eIF3c). Scoles, D.R., Yong, W.H., Qin, Y., Wawrowsky, K., Pulst, S.M. Hum. Mol. Genet. (2006)
- HRS inhibits EGF receptor signaling in the RT4 rat schwannoma cell line. Scoles, D.R., Qin, Y., Nguyen, V., Gutmann, D.H., Pulst, S.M. Biochem. Biophys. Res. Commun. (2005)
- Effects of Nf2 missense mutations on schwannomin interactions. Scoles, D.R., Chen, M., Pulst, S.M. Biochem. Biophys. Res. Commun. (2002)
- Neurofibromatosis 2 (NF2) tumor suppressor schwannomin and its interacting protein HRS regulate STAT signaling. Scoles, D.R., Nguyen, V.D., Qin, Y., Sun, C.X., Morrison, H., Gutmann, D.H., Pulst, S.M. Hum. Mol. Genet. (2002)