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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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Stefan Ehlers

Microbial Inflammation Research

Research Center Borstel

Parkallee 1

23845 Borstel

Germany

[email]@fz-borstel.de

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Microbial Inflammation Research, Research Center Borstel, Parkallee 1, 23845 Borstel, Germany. 2010
  • Molecular Inflammation Medicine, Christian-Albrechts-Universität, Kiel, Germany; Microbial Inflammation Research, Research Center Borstel, Germany. 2009 - 2010
  • Division of Molecular Infection Biology, Research Center Borstel, Germany. 1999 - 2009
  • Division of Molecular Infection Biology, Research Center Borstel, Center for Medicine and Biosciences, D-23845 Borstel, Germany. 2001 - 2005

References

  1. DC-SIGN and mannosylated surface structures of Mycobacterium tuberculosis: a deceptive liaison. Ehlers, S. Eur. J. Cell Biol. (2010) [Pubmed]
  2. TB or not TB? Fishing for Molecules Making Permissive granulomas. Ehlers, S. Cell. Host. Microbe (2010) [Pubmed]
  3. Fucosyltransferase IV and VII-directed selectin ligand function determines long-term survival in experimental tuberculosis. Ehlers, S., Schreiber, T., Dunzendorfer, A., Lowe, J.B., Hölscher, C. Immunobiology (2009) [Pubmed]
  4. Lazy, dynamic or minimally recrudescent? On the elusive nature and location of the mycobacterium responsible for latent tuberculosis. Ehlers, S. Infection (2009) [Pubmed]
  5. Why does tumor necrosis factor targeted therapy reactivate tuberculosis?. Ehlers, S. J. Rheumatol. Suppl (2005) [Pubmed]
  6. Tumor necrosis factor and its blockade in granulomatous infections: differential modes of action of infliximab and etanercept?. Ehlers, S. Clin. Infect. Dis. (2005) [Pubmed]
  7. Commentary: adaptive immunity in the absence of innate immune responses? The un-Tolled truth of the silent invaders. Ehlers, S. Eur. J. Immunol. (2004) [Pubmed]
  8. The lymphotoxin beta receptor is critically involved in controlling infections with the intracellular pathogens Mycobacterium tuberculosis and Listeria monocytogenes. Ehlers, S., Hölscher, C., Scheu, S., Tertilt, C., Hehlgans, T., Suwinski, J., Endres, R., Pfeffer, K. J. Immunol. (2003) [Pubmed]
  9. Role of tumour necrosis factor (TNF) in host defence against tuberculosis: implications for immunotherapies targeting TNF. Ehlers, S. Ann. Rheum. Dis. (2003) [Pubmed]
  10. Mycobacterium avium infection in CD14-deficient mice fails to substantiate a significant role for CD14 in antimycobacterial protection or granulomatous inflammation. Ehlers, S., Reiling, N., Gangloff, S., Woltmann, A., Goyert, S. Immunology (2001) [Pubmed]
  11. Differential requirement for interferon-gamma to restrict the growth of or eliminate some recently identified species of nontuberculous mycobacteria in vivo. Ehlers, S., Richter, E. Clin. Exp. Immunol. (2001) [Pubmed]
  12. Alphabeta T cell receptor-positive cells and interferon-gamma, but not inducible nitric oxide synthase, are critical for granuloma necrosis in a mouse model of mycobacteria-induced pulmonary immunopathology. Ehlers, S., Benini, J., Held, H.D., Roeck, C., Alber, G., Uhlig, S. J. Exp. Med. (2001) [Pubmed]
  13. Gamma interferon is essential for clearing Mycobacterium genavense infection. Ehlers, S., Richter, E. Infect. Immun. (2000) [Pubmed]
  14. Lethal granuloma disintegration in mycobacteria-infected TNFRp55-/- mice is dependent on T cells and IL-12. Ehlers, S., Kutsch, S., Ehlers, E.M., Benini, J., Pfeffer, K. J. Immunol. (2000) [Pubmed]
  15. Immunity to tuberculosis: a delicate balance between protection and pathology. Ehlers, S. FEMS Immunol. Med. Microbiol. (1999) [Pubmed]
 
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