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Yoshinori Seko

Department of Cardiovascular Medicine

Graduate School of Medicine

University of Tokyo

7-3-1 Hongo

Japan

[email]@umin.ac.jp

Name/email consistency: medium

 
 
 
 
 
 
 

Affiliations

  • Department of Cardiovascular Medicine, Graduate School of Medicine, University of Tokyo, 7-3-1 Hongo, Japan. 1998 - 2007
  • Graduate School of Medicine, University of Tokyo, Bunkyo-ku, Tokyo, Jappan. 1999

References

  1. Roles of programmed death-1 (PD-1)/PD-1 ligands pathway in the development of murine acute myocarditis caused by coxsackievirus B3. Seko, Y., Yagita, H., Okumura, K., Azuma, M., Nagai, R. Cardiovasc. Res. (2007) [Pubmed]
  2. Giant cell and Takayasu arteritis. Seko, Y. Curr. Opin. Rheumatol (2007) [Pubmed]
  3. Effect of the angiotensin II receptor blocker olmesartan on the development of murine acute myocarditis caused by coxsackievirus B3. Seko, Y. Clin. Sci. (2006) [Pubmed]
  4. Expression of costimulatory molecules (4-1BBL and Fas) and major histocompatibility class I chain-related A (MICA) in aortic tissue with Takayasu's arteritis. Seko, Y., Sugishita, K., Sato, O., Takagi, A., Tada, Y., Matsuo, H., Yagita, H., Okumura, K., Nagai, R. J. Vasc. Res. (2004) [Pubmed]
  5. Serum levels of endostatin, vascular endothelial growth factor (VEGF) and hepatocyte growth factor (HGF) in patients with acute myocardial infarction undergoing early reperfusion therapy. Seko, Y., Fukuda, S., Nagai, R. Clin. Sci. (2004) [Pubmed]
  6. Hypoxia followed by reoxygenation induces secretion of cyclophilin A from cultured rat cardiac myocytes. Seko, Y., Fujimura, T., Taka, H., Mineki, R., Murayama, K., Nagai, R. Biochem. Biophys. Res. Commun. (2004) [Pubmed]
  7. Role of Fas/FasL pathway in the activation of infiltrating cells in murine acute myocarditis caused by Coxsackievirus B3. Seko, Y., Kayagaki, N., Seino, K., Yagita, H., Okumura, K., Nagai, R. J. Am. Coll. Cardiol. (2002) [Pubmed]
  8. Expression of tumor necrosis factor ligand superfamily costimulatory molecules CD27L, CD30L, OX40L and 4-1BBL in the heart of patients with acute myocarditis and dilated cardiomyopathy. Seko, Y., Ishiyama, S., Nishikawa, T., Kasajima, T., Hiroe, M., Suzuki, S., Ishiwata, S., Kawai, S., Tanaka, Y., Azuma, M., Kobata, T., Yagita, H., Okumura, K., Nagai, R. Cardiovasc. Pathol. (2002) [Pubmed]
  9. Expression of tumour necrosis factor (TNF) ligand superfamily co-stimulatory molecules CD30L, CD27L, OX40L, and 4-1BBL in murine hearts with acute myocarditis caused by Coxsackievirus B3. Seko, Y., Takahashi, N., Oshima, H., Shimozato, O., Akiba, H., Takeda, K., Kobata, T., Yagita, H., Okumura, K., Azuma, M., Nagai, R. J. Pathol. (2001) [Pubmed]
  10. Restricted usage of T-cell receptor Vgamma-Vdelta genes and expression of costimulatory molecules in Takayasu's arteritis. Seko, Y., Takahashi, N., Tada, Y., Yagita, H., Okumura, K., Nagai, R. Int. J. Cardiol. (2000) [Pubmed]
  11. Pulsatile stretch activates mitogen-activated protein kinase (MAPK) family members and focal adhesion kinase (p125(FAK)) in cultured rat cardiac myocytes. Seko, Y., Takahashi, N., Tobe, K., Kadowaki, T., Yazaki, Y. Biochem. Biophys. Res. Commun. (1999) [Pubmed]
  12. Effects of in vivo administration of anti-B7-1/B7-2 monoclonal antibodies on the survival of mice with chronic ongoing myocarditis caused by Coxsackievirus B3. Seko, Y., Takahashi, N., Yagita, H., Okumura, K., Azuma, M., Yazaki, Y. J. Pathol. (1999) [Pubmed]
  13. Expression of tumour necrosis factor (TNF) receptor/ligand superfamily co-stimulatory molecules CD40, CD30L, CD27L, and OX40L in murine hearts with chronic ongoing myocarditis caused by coxsackie virus B3. Seko, Y., Takahashi, N., Oshima, H., Shimozato, O., Akiba, H., Kobata, T., Yagita, H., Okumura, K., Azuma, M., Yazaki, Y. J. Pathol. (1999) [Pubmed]
  14. Pulsatile stretch stimulates vascular endothelial growth factor (VEGF) secretion by cultured rat cardiac myocytes. Seko, Y., Seko, Y., Takahashi, N., Shibuya, M., Yazaki, Y. Biochem. Biophys. Res. Commun. (1999) [Pubmed]
  15. Expression of costimulatory molecule CD40 in murine heart with acute myocarditis and reduction of inflammation by treatment with anti-CD40L/B7-1 monoclonal antibodies. Seko, Y., Takahashi, N., Azuma, M., Yagita, H., Okumura, K., Yazaki, Y. Circ. Res. (1998) [Pubmed]
 
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