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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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Seung K. Kim

Departments of Developmental Biology and Medicine (Oncology Division)

Stanford University School of Medicine

Stanford

California 94305

USA

[email]@*.stanford.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Departments of Developmental Biology and Medicine (Oncology Division), Stanford University School of Medicine, Stanford, California 94305, USA. 2000 - 2006
  • Beckman Center Room B300, Mail Stop 5329, Stanford University, 279 Campus Drive, USA. 2002
  • Howard Hughes Medical Institute and Department of Molecular and Cellular Biology, Harvard University, Cambridge, MA 02138, USA. 1998

References

  1. Intrinsic regulators of pancreatic beta-cell proliferation. Heit, J.J., Karnik, S.K., Kim, S.K. Annu. Rev. Cell Dev. Biol. (2006) [Pubmed]
  2. Conserved mechanisms of glucose sensing and regulation by Drosophila corpora cardiaca cells. Kim, S.K., Rulifson, E.J. Nature (2004) [Pubmed]
  3. Pbx1 inactivation disrupts pancreas development and in Ipf1-deficient mice promotes diabetes mellitus. Kim, S.K., Selleri, L., Lee, J.S., Zhang, A.Y., Gu, X., Jacobs, Y., Cleary, M.L. Nat. Genet. (2002) [Pubmed]
  4. Pancreatic islet cell replacement: successes and opportunities. Kim, S.K. Ann. N. Y. Acad. Sci. (2002) [Pubmed]
  5. Intercellular signals regulating pancreas development and function. Kim, S.K., Hebrok, M. Genes Dev. (2001) [Pubmed]
  6. Activin receptor patterning of foregut organogenesis. Kim, S.K., Hebrok, M., Li, E., Oh, S.P., Schrewe, H., Harmon, E.B., Lee, J.S., Melton, D.A. Genes Dev. (2000) [Pubmed]
  7. Pancreas development is promoted by cyclopamine, a hedgehog signaling inhibitor. Kim, S.K., Melton, D.A. Proc. Natl. Acad. Sci. U.S.A. (1998) [Pubmed]
 
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