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Bronislaw L. Slomiany

UMDNJ-NJ Dental School

Research Center

C875, University of Medicine and Dentistry of New Jersey

110 Bergen Street

USA

[email]@umdnj.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • UMDNJ-NJ Dental School, Research Center, C875, University of Medicine and Dentistry of New Jersey, 110 Bergen Street, USA. 1999 - 2011
  • Research Center, C875, UMDJN-NJ Dental School, 110 Bergen Street, USA. 2005 - 2006

References

  1. Role of ghrelin-induced cSrc activation in modulation of gastric mucosal inflammatory responses to Helicobacter pylori. Slomiany, B.L., Slomiany, A. Inflammopharmacology (2011) [Pubmed]
  2. Ghrelin suppression of Helicobacter pylori-induced S-nitrosylation-dependent Akt inactivation exerts modulatory influence on gastric mucin synthesis. Slomiany, B.L., Slomiany, A. Inflammopharmacology (2011) [Pubmed]
  3. Constitutive nitric oxide synthase-mediated caspase-3 S-nitrosylation in ghrelin protection against Porphyromonas gingivalis-induced salivary gland acinar cell apoptosis. Slomiany, B.L., Slomiany, A. Inflammopharmacology (2010) [Pubmed]
  4. Role of constitutive nitric oxide synthase S-nitrosylation in Helicobacter pylori-induced gastric mucosal cell apoptosis: effect of ghrelin. Slomiany, B.L., Slomiany, A. Inflammopharmacology (2010) [Pubmed]
  5. Ghrelin protection against lipopolysaccharide-induced gastric mucosal cell apoptosis involves constitutive nitric oxide synthase-mediated caspase-3 S-nitrosylation. Slomiany, B.L., Slomiany, A. Mediators Inflamm. (2010) [Pubmed]
  6. Involvement of constitutive nitric oxide synthase in ghrelin-induced cytosolic phospholipase A(2) activation in gastric mucosal cell protection against ethanol cytotoxicity. Slomiany, B.L., Slomiany, A. Inflammopharmacology (2009) [Pubmed]
  7. Role of epidermal growth factor receptor transactivation in the activation of cytosolic phospholipase A(2) in leptin protection of salivary gland acinar cells against ethanol cytotoxicity. Slomiany, B.L., Slomiany, A. J. Physiol. Pharmacol. (2009) [Pubmed]
  8. Leptin-induced cytosolic phospholipase A2 activation in gastric mucosal protection against ethanol cytotoxicity involves epidermal growth factor receptor transactivation. Slomiany, B.L., Slomiany, A. Inflammopharmacology (2009) [Pubmed]
  9. Leptin protection of salivary gland acinar cells against ethanol cytotoxicity involves Src kinase-mediated parallel activation of prostaglandin and constitutive nitric oxide synthase pathways. Slomiany, B.L., Slomiany, A. Inflammopharmacology (2008) [Pubmed]
  10. Src kinase-mediated parallel activation of prostaglandin and constitutive nitric oxide synthase pathways in leptin protection of gastric mucosa against ethanol cytotoxicity. Slomiany, B.L., Slomiany, A. J. Physiol. Pharmacol. (2008) [Pubmed]
  11. Interference by leptin with Helicobacter pylori lipopolysaccharide-induced cytosolic phospholipase A2 activation in gastric mucosal cells. Slomiany, B.L., Slomiany, A. J. Physiol. Pharmacol. (2007) [Pubmed]
  12. Cytosolic phospholipase A2 activation in Helicobacter pylori lipopolysaccharide-induced interference with gastric mucin synthesis. Slomiany, B.L., Slomiany, A. IUBMB. Life (2006) [Pubmed]
  13. Porphyromonas gingivalis lipopolysaccharide-induced cytosolic phospholipase A2 activation interferes with salivary mucin synthesis via platelet activating factor generation. Slomiany, B.L., Slomiany, A. Inflammopharmacology (2006) [Pubmed]
  14. Leptin modulates the detrimental effect of Porphyromonas gingivalis lipopolysaccharide-induced cytosolic phospholipase A2 activation on salivary mucin synthesis via ERK-signal transduction. Slomiany, B.L., Slomiany, A. Inflammopharmacology (2006) [Pubmed]
  15. Gastric mucin secretion in response to beta-adrenergic G protein-coupled receptor activation is mediated by SRC kinase-dependent epidermal growth factor receptor transactivation. Słomiany, B.L., Słomiany, A. J. Physiol. Pharmacol. (2005) [Pubmed]
  16. Role of leptin in modulation of Porphyromonas gingivalis lipopolysaccharide-induced up-regulation of endothelin-1 in salivary gland acinar cells. Slomiany, B.L., Slomiany, A. IUBMB. Life (2005) [Pubmed]
  17. Endothelin-1-dependent leptin induction in gastric mucosal inflammatory responses to Helicobacter pylori lipopolysaccharide. Slomiany, B.L., Slomiany, A. Biochem. Biophys. Res. Commun. (2005) [Pubmed]
  18. Up-regulation in endothelin-1 by Helicobacter pylori lipopolysaccharide interferes with gastric mucin synthesis via epidermal growth factor receptor transactivation. Slomiany, B.L., Slomiany, A. Scand. J. Gastroenterol. (2005) [Pubmed]
  19. Endothelin-1-dependent up-regulation of leptin production in gastric mucosal injury by indomethacin. Slomiany, B.L., Slomiany, A. Inflammopharmacology (2005) [Pubmed]
  20. Role of endothelin-1-dependent up-regulation of leptin in oral mucosal repair. Slomiany, B.L., Slomiany, A. J. Physiol. Pharmacol. (2005) [Pubmed]
  21. Platelet-activating factor mediates Helicobacter pylori lipopolysaccharide interference with gastric mucin synthesis. Slomiany, B.L., Slomiany, A. IUBMB. Life (2004) [Pubmed]
  22. Platelet-activating factor mediates Porphyromonas gingivalis lipopolysaccharide interference with salivary mucin synthesis via phosphatidylinositol 3-kinase-dependent constitutive nitric-oxide synthase activation. Slomiany, B.L., Slomiany, A. J. Physiol. Pharmacol. (2004) [Pubmed]
  23. Salivary phospholipid secretion in response to beta-adrenergic stimulation is mediated by Src kinase-dependent epidermal growth factor receptor transactivation. Slomiany, B.L., Slomiany, A. Biochem. Biophys. Res. Commun. (2004) [Pubmed]
  24. Src kinase-dependent epidermal growth factor receptor transactivation in PPARgamma ligand-induced suppression of Porphyromonas gingivalis interference with salivary mucin synthesis. Slomiany, B.L., Slomiany, A. IUBMB. Life (2004) [Pubmed]
  25. Role of epidermal growth factor receptor transactivation in PPAR gamma-dependent suppression of Helicobacter pylori interference with gastric mucin synthesis. Slomiany, B.L., Slomiany, A. Inflammopharmacology (2004) [Pubmed]
  26. Secretion of gastric mucus phospholipids in response to beta-adrenergic G protein-coupled receptor activation is mediated by SRC kinase-dependent epidermal growth factor receptor transactivation. Slomiany, B.L., Slomiany, A. J. Physiol. Pharmacol. (2004) [Pubmed]
  27. Src-kinase-dependent epidermal growth factor receptor transactivation in salivary mucin secretion in response to beta-adrenergic G-protein-coupled receptor activation. Slomiany, B.L., Slomiany, A. Inflammopharmacology (2004) [Pubmed]
  28. Porphyromonas gingivalis lipopolysaccharide-induced up-regulation in endothelin-1 interferes with salivary mucin synthesis via epidermal growth factor receptor transactivation. Slomiany, B.L., Slomiany, A. IUBMB. Life (2004) [Pubmed]
  29. Activation of peroxisome proliferator-activated receptor gamma impedes Porphyromonas gingivalis lipopolysaccharide interference with salivary mucin synthesis through phosphatidylinositol 3-kinase/erk pathway. Slomiany, B.L., Slomiany, A. J. Physiol. Pharmacol. (2003) [Pubmed]
  30. Impedance of Helicobacter pylori lipopolysaccharide interference with gastric mucin synthesis by peroxisome proliferator-activated receptor gamma activation involves phosphatidylinositol 3-kinase/ERK pathway. Slomiany, B.L., Slomiany, A. IUBMB. Life (2003) [Pubmed]
  31. Platelet-activating factor modulates gastric mucosal inflammatory responses to Helicobacter pylori lipopolysaccharide. Slomiany, B.L., Slomiany, A. Biochem. Biophys. Res. Commun. (2003) [Pubmed]
  32. Biphasic role of platelet-activating factor in oral mucosal ulcer healing. Slomiany, B.L., Slomiany, A. IUBMB. Life (2003) [Pubmed]
  33. Leptin suppresses Porphyromonas gingivalis lipopolysaccharide interference with salivary mucin synthesis. Slomiany, B.L., Slomiany, A. Biochem. Biophys. Res. Commun. (2003) [Pubmed]
  34. Peroxisome proliferator-activated receptor gamma activation counters the detrimental effect of Helicobacter pylori lipopolysaccharide on gastric mucin synthesis. Slomiany, B.L., Slomiany, A. Inflammopharmacology (2003) [Pubmed]
  35. Disruption in gastric mucin synthesis by Helicobacter pylori lipopolysaccharide involves ERK and p38 mitogen-activated protein kinase participation. Slomiany, B.L., Slomiany, A. Biochem. Biophys. Res. Commun. (2002) [Pubmed]
  36. Activation of peroxisome proliferator-activated receptor gamma suppresses inducible cyclooxygenase and nitric oxide synthase during oral mucosal ulcer healing. Slomiany, B.L., Slomiany, A. J. Physiol. Pharmacol. (2002) [Pubmed]
  37. Nitric oxide interferes with salivary mucin synthesis: involvement of ERK and p38 mitogen-activated protein kinase. Slomiany, B.L., Slomiany, A. J. Physiol. Pharmacol. (2002) [Pubmed]
  38. Porphyromonas gingivalis lipopolysaccharide interferes with salivary mucin synthesis through inducible nitric oxide synthase activation by ERK and p38 kinase. Slomiany, B.L., Slomiany, A. Biochem. Biophys. Res. Commun. (2002) [Pubmed]
  39. Nitric oxide as a modulator of gastric mucin synthesis: role of ERK and p38 mitogen-activated protein kinase activation. Slomiany, B.L., Slomiany, A. IUBMB. Life (2002) [Pubmed]
  40. Suppression of gastric mucosal inflammatory responses to Helicobacter pylori lipopolysaccharide by peroxisome proliferator-activated receptor gamma activation. Slomiany, B.L., Slomiany, A. IUBMB. Life (2002) [Pubmed]
  41. Nonsteroidal anti-inflammatory drugs impair oral mucosal repair by eliciting disturbances in endothelin-converting enzyme-1 and constitutive nitric oxide synthase. Slomiany, B.L., Slomiany, A. J. Physiol. Pharmacol. (2001) [Pubmed]
  42. Up-regulation of gastric mucosal inflammatory responses to Helicobacter pylori lipopolysaccharide by aspirin but not indomethacin. Slomiany, B.L., Piotrowski, J., Slomiany, A. J. Endotoxin Res. (2001) [Pubmed]
  43. Role of ERK and p38 mitogen-activated protein kinase cascades in gastric mucosal inflammatory responses to Helicobacter pylori lipopolysaccharide. Slomiany, B.L., Slomiany, A. IUBMB. Life (2001) [Pubmed]
  44. Up-regulation of endothelin-converting enzyme-1 in gastric mucosal inflammatory responses to Helicobacter pylori lipopolysaccharide. Slomiany, B.L., Piotrowski, J., Slomiany, A. Biochem. Biophys. Res. Commun. (2000) [Pubmed]
  45. Induction of endothelin-converting enzyme-1 in gastric mucosal injury by idomethacin. Slomiany, B.L., Slomiany, A. Biochem. Biophys. Res. Commun. (2000) [Pubmed]
  46. Role of interleukin-4 in down-regulation of endothelin-1 during gastric ulcer healing: effect of sucralfate. Slomiany, B.L., Piotrowski, J., Slomiany, A. J. Physiol. Pharmacol. (2000) [Pubmed]
  47. Suppression of endothelin-converting enzyme-1 during buccal mucosal ulcer healing: effect of chronic alcohol ingestion. Slomiany, B.L., Piotrowski, J., Slomiany, A. Biochem. Biophys. Res. Commun. (2000) [Pubmed]
  48. Up-regulation of endothelin-1 in gastric mucosal inflammatory responses to Helicobacter pylori Lipopolysaccharide: effect of omeprazole and sucralfate. Slomiany, B.L., Piotrowski, J., Slomiany, A. J. Physiol. Pharmacol. (2000) [Pubmed]
  49. Omeprazole fails to suppress up-regulation of gastric mucosal endothelin-converting enzyme-1 by Helicobacter pylori lipopolysaccharide. Slomiany, B.L., Piotrowski, J., Slomiany, A. J. Physiol. Pharmacol. (2000) [Pubmed]
  50. Aspirin ingestion impairs oral mucosal ulcer healing by inducing membrane-bound tumor necrosis factor-alpha release. Slomiany, B.L., Slomiany, A. IUBMB. Life (2000) [Pubmed]
  51. Involvement of endothelin-1 in up-regulation of gastric mucosal inflammatory responses to Helicobacter pylori lipopolysaccharide. Slomiany, B.L., Piotrowski, J., Slomiany, A. Biochem. Biophys. Res. Commun. (1999) [Pubmed]
  52. Effect of ebrotidine on gastric mucosal inflammatory responses to Helicobacter pylori lipopolysaccharide. Slomiany, B.L., Piotrowski, J., Slomiany, A. J. Physiol. Pharmacol. (1999) [Pubmed]
  53. Suppression of caspase-3 and nitric-oxide synthase-2 during buccal mucosal ulcer healing: effect of chronic alcohol ingestion. Slomiany, B.L., Piotrowski, J., Slomiany, A. IUBMB. Life (1999) [Pubmed]
  54. Gastric mucosal inflammatory responses toHelicobacter pylori lipopolysaccharide: suppression of caspase-3 and nitric oxide synthase-2 by omeprazole and sucralfate. Slomiany, B.L., Piotrowski, J., Slomiany, A. Inflammopharmacology (1999) [Pubmed]
 
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