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Sidhartha D. Ray

Division ofPharmaceutical Sciences

Arnold & Marie Schwartz College of Pharmacy and Health Sciences

Long Island University

Brooklyn

USA

[email]@liu.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Division ofPharmaceutical Sciences, Arnold & Marie Schwartz College of Pharmacy and Health Sciences, Long Island University, Brooklyn, USA. 2000 - 2008
  • Molecular Toxicology Program, Department of Pharmacology, Toxicology and Medicinal Chemistry, Long Island University, USA. 2001

References

  1. Synergy of Pancratistatin and Tamoxifen in inducing apoptosis in breast cancer cells. Ray, S. Cancer Biol. Ther. (2008) [Pubmed]
  2. Long term exposure effect of a unique metabolic nutrition system containing a diverse group of phytochemicals on serum chemistry and genomic and non-genomic changes in the liver of female B6C3F1 mice. Ray, S.D., Parmar, M., Syed, I., Rathod, J., Zinkovsky, D., Bulku, E., Gigliotti, J., Hackman, R.M., Stohs, S.J. Phytother. Res (2008) [Pubmed]
  3. Pre-exposure to a novel nutritional mixture containing a series of phytochemicals prevents acetaminophen-induced programmed and unprogrammed cell deaths by enhancing BCL-XL expression and minimizing oxidative stress in the liver. Ray, S.D., Patel, N., Shah, N., Nagori, A., Naqvi, A., Stohs, S.J. Mol. Cell. Biochem. (2006) [Pubmed]
  4. Short-term and long-term in vivo exposure to an ephedra- and caffeine-containing metabolic nutrition system does not induce cardiotoxicity in B6C3F1 mice. Ray, S., Phadke, S., Patel, C., Hackman, R.M., Stohs, S. Arch. Toxicol. (2005) [Pubmed]
  5. Proanthocyanidin exposure to B6C3F1 mice significantly attenuates dimethylnitrosamine-induced liver tumor induction and mortality by differentially modulating programmed and unprogrammed cell deaths. Ray, S.D., Parikh, H., Bagchi, D. Mutat. Res. (2005) [Pubmed]
  6. Oxidative stress is the master operator of drug and chemically-induced programmed and unprogrammed cell death: Implications of natural antioxidants in vivo. Ray, S.D., Lam, T.S., Rotollo, J.A., Phadke, S., Patel, C., Dontabhaktuni, A., Mohammad, S., Lee, H., Strika, S., Dobrogowska, A., Bruculeri, C., Chou, A., Patel, S., Patel, R., Manolas, T., Stohs, S. Biofactors (2004) [Pubmed]
  7. Differential effects of IH636 grape seed proanthocyanidin extract and a DNA repair modulator 4-aminobenzamide on liver microsomal cytochrome 4502E1-dependent aniline hydroxylation. Ray, S.D., Parikh, H., Hickey, E., Bagchi, M., Bagchi, D. Mol. Cell. Biochem. (2001) [Pubmed]
  8. Ca(2+)-calmodulin antagonist chlorpromazine and poly(ADP-ribose) polymerase modulators 4-aminobenzamide and nicotinamide influence hepatic expression of BCL-XL and P53 and protect against acetaminophen-induced programmed and unprogrammed cell death in mice. Ray, S.D., Balasubramanian, G., Bagchi, D., Reddy, C.S. Free Radic. Biol. Med. (2001) [Pubmed]
  9. Unique organoprotective properties of a novel IH636 grape seed proanthocyanidin extract on cadmium chloride-induced nephrotoxicity, dimethylnitrosamine (DMN)-induced splenotoxicity and mocap-induced neurotoxicity in mice. Ray, S.D., Wong, V., Rinkovsky, A., Bagchi, M., Raje, R.R., Bagchi, D. Res. Commun. Mol. Pathol. Pharmacol. (2000) [Pubmed]
  10. In vivo protection of dna damage associated apoptotic and necrotic cell deaths during acetaminophen-induced nephrotoxicity, amiodarone-induced lung toxicity and doxorubicin-induced cardiotoxicity by a novel IH636 grape seed proanthocyanidin extract. Ray, S.D., Patel, D., Wong, V., Bagchi, D. Res. Commun. Mol. Pathol. Pharmacol. (2000) [Pubmed]
 
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