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Shi Yong Sun

Department of Immunology

The University of Texas MD Anderson Cancer Center

7455 Fannin Street

Box 902

USA

[email]@mdanderson.org

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Department of Immunology, The University of Texas MD Anderson Cancer Center, 7455 Fannin Street, Box 902, USA. 2000 - 2011
  • Department of Immunology, The University of Texas MD Anderson Cancer Center and The University of Texas Graduate School of Biomedical Sciences at Houston, Houston, TX, 77030, USA. 2011
  • Department of Immunology, MD Anderson Cancer Center, University of Texas, Houston, USA. 2008

References

  1. Mutual regulation between deubiquitinase CYLD and retroviral oncoprotein Tax. Wu, X., Zhang, M., Sun, S.C. Cell. Biosci (2011) [Pubmed]
  2. NF-κB as a Target for Oncogenic Viruses. Sun, S.C., Cesarman, E. Curr. Top. Microbiol. Immunol. (2011) [Pubmed]
  3. Controlling the fate of NIK: a central stage in noncanonical NF-kappaB signaling. Sun, S.C. Sci. Signal (2010) [Pubmed]
  4. CYLD: a tumor suppressor deubiquitinase regulating NF-kappaB activation and diverse biological processes. Sun, S.C. Cell Death Differ. (2010) [Pubmed]
  5. New insights into NF-kappaB regulation and function. Sun, S.C., Ley, S.C. Trends Immunol. (2008) [Pubmed]
  6. Deubiquitylation and regulation of the immune response. Sun, S.C. Nat. Rev. Immunol. (2008) [Pubmed]
  7. The synthetic retinoid CD437 selectively induces apoptosis in human lung cancer cells while sparing normal human lung epithelial cells. Sun, S.Y., Yue, P., Chen, X., Hong, W.K., Lotan, R. Cancer Res. (2002) [Pubmed]
  8. How much do we know about retinoid-regulated genes?. Sun, S.Y. Cancer Biol. Ther. (2002) [Pubmed]
  9. Identification of retinamides that are more potent than N-(4-hydroxyphenyl)retinamide in inhibiting growth and inducing apoptosis of human head and neck and lung cancer cells. Sun, S.Y., Yue, P., Kelloff, G.J., Steele, V.E., Lippman, S.M., Hong, W.K., Lotan, R. Cancer Epidemiol. Biomarkers Prev. (2001) [Pubmed]
  10. Augmentation of tumor necrosis factor-related apoptosis-inducing ligand (TRAIL)-induced apoptosis by the synthetic retinoid 6-[3-(1-adamantyl)-4-hydroxyphenyl]-2-naphthalene carboxylic acid (CD437) through up-regulation of TRAIL receptors in human lung cancer cells. Sun, S.Y., Yue, P., Hong, W.K., Lotan, R. Cancer Res. (2000) [Pubmed]
 
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