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Stefan Müller

Department of Clinical Research

University of Bern

CH-3010 Bern

Switzerland

[email]@dkf.unibe.ch

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Department of Clinical Research, University of Bern, CH-3010 Bern, Switzerland. 2010
  • Institute of Pathology, Division of Experimental Pathology, University of Bern, Switzerland. 2009
  • Department of Clinical Research, Division of Gastroenterology, University Hospital, University of Bern, Switzerland. 2006
  • Division of Gastroenterology, Department of Clinical Research, University Hospital Bern. 2005

References

  1. Mannan-binding lectin deficiency results in unusual antibody production and excessive experimental colitis in response to mannose-expressing mild gut pathogens. Müller, S., Schaffer, T., Flogerzi, B., Seibold-Schmid, B., Schnider, J., Takahashi, K., Darfeuille-Michaud, A., Vazeille, E., Schoepfer, A.M., Seibold, F. Gut (2010) [Pubmed]
  2. Soluble TNF-alpha but not transmembrane TNF-alpha sensitizes T cells for enhanced activation-induced cell death. Müller, S., Rihs, S., Schneider, J.M., Paredes, B.E., Seibold, I., Brunner, T., Mueller, C. Eur. J. Immunol. (2009) [Pubmed]
  3. Galectin-3 modulates T cell activity and is reduced in the inflamed intestinal epithelium in IBD. Müller, S., Schaffer, T., Flogerzi, B., Fleetwood, A., Weimann, R., Schoepfer, A.M., Seibold, F. Inflamm. Bowel Dis. (2006) [Pubmed]
  4. Anti-Saccharomyces cerevisiae antibody titers are stable over time in Crohn's patients and are not inducible in murine models of colitis. Müller, S., Styner, M., Seibold-Schmid, B., Flogerzi, B., Mähler, M., Konrad, A., Seibold, F. World J. Gastroenterol. (2005) [Pubmed]
 
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