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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)

Andrea Steinborn

Department of Obstetrics and Gynaecology

J.-W. Goethe-University of Frankfurt



Name/email consistency: high



  • Department of Obstetrics and Gynaecology, J.-W. Goethe-University of Frankfurt, Germany. 1999 - 2004


  1. Anti-fetal immune response mechanisms may be involved in the pathogenesis of placental abruption. Steinborn, A., Seidl, C., Sayehli, C., Sohn, C., Seifried, E., Kaufmann, M., Schmitt, E. Clin. Immunol. (2004) [Pubmed]
  2. Soluble HLA-DR levels in the maternal circulation of normal and pathologic pregnancy. Steinborn, A., Rebmann, V., Scharf, A., Sohn, C., Grosse-Wilde, H. Am. J. Obstet. Gynecol. (2003) [Pubmed]
  3. Placental abruption is associated with decreased maternal plasma levels of soluble HLA-G. Steinborn, A., Rebmann, V., Scharf, A., Sohn, C., Grosse-Wilde, H. J. Clin. Immunol. (2003) [Pubmed]
  4. Serum intercellular adhesion molecule-1 levels and histologic chorioamnionitis. Steinborn, A., Sohn, C., Scharf, A., Geka, F., Heger, S., Kaufmann, M. Obstet. Gynecol (2000) [Pubmed]
  5. Spontaneous labour at term is associated with fetal monocyte activation. Steinborn, A., Sohn, C., Sayehli, C., Baudendistel, A., Hüwelmeier, D., Solbach, C., Schmitt, E., Kaufmann, M. Clin. Exp. Immunol. (1999) [Pubmed]
  6. Labour-associated expression of intercellular adhesion molecule-1 (ICAM-1) in placental endothelial cells indicates participation of immunological processes in parturition. Steinborn, A., Sohn, C., Heger, S., Niederhut, A., Hildenbrand, R., Kaufmann, M. Placenta (1999) [Pubmed]
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