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Toshio Miyata

Institute of Medical Sciences and Division of Nephrology

Hypertension and Metabolism

Tokai University School of Medicine

Kanagawa

Japan

[email]@is.icc.u-tokai.ac.jp

Name/email consistency: high

 
 
 
 
 
 
 

Affiliation

  • Institute of Medical Sciences and Division of Nephrology, Hypertension and Metabolism, Tokai University School of Medicine, Kanagawa, Japan. 1998 - 2008

References

  1. Inhibition of advanced glycation end products: an implicit goal in clinical medicine for the treatment of diabetic nephropathy? Miyata, T., Izuhara, Y. Ann. N. Y. Acad. Sci. (2008) [Pubmed]
  2. Toward better renoprotection: lessons from angiotensin receptor blockers. Miyata, T., Takizawa, S. Hemodial. Int (2007) [Pubmed]
  3. Serpinopathy and endoplasmic reticulum stress. Miyata, T., Inagi, R., Sugiyama, S., Usuda, N. Med. Mol. Morphol (2005) [Pubmed]
  4. From molecular footprints of disease to new therapeutic interventions in diabetic nephropathy. Miyata, T., Yamamoto, M., Izuhara, Y. Ann. N. Y. Acad. Sci. (2005) [Pubmed]
  5. From molecular footprints of disease to new therapeutic interventions in diabetic nephropathy: a detective story. Miyata, T., Kurokawa, K., van Ypersele de Strihou, C. Curr. Drug Targets Immune Endocr. Metabol. Disord. (2005) [Pubmed]
  6. Angiotensin II receptor blockers and angiotensin converting enzyme inhibitors: implication of radical scavenging and transition metal chelation in inhibition of advanced glycation end product formation. Miyata, T., van Ypersele de Strihou, C. Arch. Biochem. Biophys. (2003) [Pubmed]
  7. Carbonyl stress and diabetic complications. Miyata, T., Ishikawa, N., van Ypersele de Strihou, C. Clin. Chem. Lab. Med. (2003) [Pubmed]
  8. Toward better dialysis compatibility: advances in the biochemistry and pathophysiology of the peritoneal membranes. Miyata, T., Devuyst, O., Kurokawa, K., van Ypersele de Strihou, C. Kidney Int. (2002) [Pubmed]
  9. Overexpression of the serpin megsin induces progressive mesangial cell proliferation and expansion. Miyata, T., Inagi, R., Nangaku, M., Imasawa, T., Sato, M., Izuhara, Y., Suzuki, D., Yoshino, A., Onogi, H., Kimura, M., Sugiyama, S., Kurokawa, K. J. Clin. Invest. (2002) [Pubmed]
  10. Angiotensin II receptor antagonists and angiotensin-converting enzyme inhibitors lower in vitro the formation of advanced glycation end products: biochemical mechanisms. Miyata, T., van Ypersele de Strihou, C., Ueda, Y., Ichimori, K., Inagi, R., Onogi, H., Ishikawa, N., Nangaku, M., Kurokawa, K. J. Am. Soc. Nephrol. (2002) [Pubmed]
  11. Reactive carbonyl compounds related uremic toxicity ("carbonyl stress"). Miyata, T., Sugiyama, S., Saito, A., Kurokawa, K. Kidney Int. Suppl. (2001) [Pubmed]
  12. Reactive carbonyl compounds as uremic toxins. Miyata, T., Akhand, A.A., Kurokawa, K., Nakashima, I. Contrib. Nephrol (2001) [Pubmed]
  13. Glyoxalase I deficiency is associated with an unusual level of advanced glycation end products in a hemodialysis patient. Miyata, T., van Ypersele de Strihou, C., Imasawa, T., Yoshino, A., Ueda, Y., Ogura, H., Kominami, K., Onogi, H., Inagi, R., Nangaku, M., Kurokawa, K. Kidney Int. (2001) [Pubmed]
  14. Relevance of oxidative and carbonyl stress to long-term uremic complications. Miyata, T., Kurokawa, K., van Ypersele de Strihou, C. Kidney Int. Suppl. (2000) [Pubmed]
  15. Mechanism of the inhibitory effect of OPB-9195 [(+/-)-2-isopropylidenehydrazono-4-oxo-thiazolidin-5-yla cetanilide] on advanced glycation end product and advanced lipoxidation end product formation. Miyata, T., Ueda, Y., Asahi, K., Izuhara, Y., Inagi, R., Saito, A., Van Ypersele De Strihou, C., Kurokawa, K. J. Am. Soc. Nephrol. (2000) [Pubmed]
  16. Advanced glycation and lipoxidation end products: role of reactive carbonyl compounds generated during carbohydrate and lipid metabolism. Miyata, T., Kurokawa, K., Van Ypersele De Strihou, C. J. Am. Soc. Nephrol. (2000) [Pubmed]
  17. 2-Isopropylidenehydrazono-4-oxo-thiazolidin-5-ylacetanilide (OPB-9195) treatment inhibits the development of intimal thickening after balloon injury of rat carotid artery: role of glycoxidation and lipoxidation reactions in vascular tissue damage. Miyata, T., Ishikawa, S., Asahi, K., Inagi, R., Suzuki, D., Horie, K., Tatsumi, K., Kurokawa, K. FEBS Lett. (1999) [Pubmed]
  18. Apolipoprotein E2/E5 variants in lipoprotein glomerulopathy recurred in transplanted kidney. Miyata, T., Sugiyama, S., Nangaku, M., Suzuki, D., Uragami, K., Inagi, R., Sakai, H., Kurokawa, K. J. Am. Soc. Nephrol. (1999) [Pubmed]
  19. Alterations in nonenzymatic biochemistry in uremia: origin and significance of "carbonyl stress" in long-term uremic complications. Miyata, T., van Ypersele de Strihou, C., Kurokawa, K., Baynes, J.W. Kidney Int. (1999) [Pubmed]
  20. A mesangium-predominant gene, megsin, is a new serpin upregulated in IgA nephropathy. Miyata, T., Nangaku, M., Suzuki, D., Inagi, R., Uragami, K., Sakai, H., Okubo, K., Kurokawa, K. J. Clin. Invest. (1998) [Pubmed]
  21. Autoxidation products of both carbohydrates and lipids are increased in uremic plasma: is there oxidative stress in uremia?. Miyata, T., Fu, M.X., Kurokawa, K., van Ypersele de Strihou, C., Thorpe, S.R., Baynes, J.W. Kidney Int. (1998) [Pubmed]
  22. Generation of protein carbonyls by glycoxidation and lipoxidation reactions with autoxidation products of ascorbic acid and polyunsaturated fatty acids. Miyata, T., Inagi, R., Asahi, K., Yamada, Y., Horie, K., Sakai, H., Uchida, K., Kurokawa, K. FEBS Lett. (1998) [Pubmed]
 
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