Anthony P. Goldstone
Department of Endocrinology
St Bartholomew's Hospital
West Smithfield
London EC1A 7BE
UK
Name/email consistency: high
- Fasting and postprandial hyperghrelinemia in Prader-Willi syndrome is partially explained by hypoinsulinemia, and is not due to peptide YY3-36 deficiency or seen in hypothalamic obesity due to craniopharyngioma. Goldstone, A.P., Patterson, M., Kalingag, N., Ghatei, M.A., Brynes, A.E., Bloom, S.R., Grossman, A.B., Korbonits, M. J. Clin. Endocrinol. Metab. (2005)
- Prader-Willi syndrome: advances in genetics, pathophysiology and treatment. Goldstone, A.P. Trends Endocrinol. Metab. (2004)
- Elevated fasting plasma ghrelin in prader-willi syndrome adults is not solely explained by their reduced visceral adiposity and insulin resistance. Goldstone, A.P., Thomas, E.L., Brynes, A.E., Castroman, G., Edwards, R., Ghatei, M.A., Frost, G., Holland, A.J., Grossman, A.B., Korbonits, M., Bloom, S.R., Bell, J.D. J. Clin. Endocrinol. Metab. (2004)
- Hypothalamic growth hormone-releasing hormone (GHRH) cell number is increased in human illness, but is not reduced in Prader-Willi syndrome or obesity. Goldstone, A.P., Unmehopa, U.A., Swaab, D.F. Clin. Endocrinol. (Oxf) (2003)
- Hypothalamic NPY and agouti-related protein are increased in human illness but not in Prader-Willi syndrome and other obese subjects. Goldstone, A.P., Unmehopa, U.A., Bloom, S.R., Swaab, D.F. J. Clin. Endocrinol. Metab. (2002)
- Leptin prevents the fall in plasma osteocalcin during starvation in male mice. Goldstone, A.P., Howard, J.K., Lord, G.M., Ghatei, M.A., Gardiner, J.V., Wang, Z.L., Wang, R.M., Girgis, S.I., Bailey, C.J., Bloom, S.R. Biochem. Biophys. Res. Commun. (2002)
- Visceral adipose tissue and metabolic complications of obesity are reduced in Prader-Willi syndrome female adults: evidence for novel influences on body fat distribution. Goldstone, A.P., Thomas, E.L., Brynes, A.E., Bell, J.D., Frost, G., Saeed, N., Hajnal, J.V., Howard, J.K., Holland, A., Bloom, S.R. J. Clin. Endocrinol. Metab. (2001)
