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Thomas Walther

Research Laboratories of Schering AG

Experimental Oncology

13342 Berlin

Germany

[email]@*.fu-berlin.de

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Research Laboratories of Schering AG, Experimental Oncology, 13342 Berlin, Germany. 2003
  • Department of Cardiology and Pneumology, University Hospital Benjamin Franklin, Free University of Berlin, Hindenburgdamm 30, Germany. 2003
  • Max-Planck-Institute for Neurological Research, Department of Experimental Neurology, Cologne, Germany. 2002
  • Department of Cardiology and Pneumology, Free University of Berlin, Berlin, Germany. 2002

References

  1. Differential regulation of in vivo angiogenesis by angiotensin II receptors. Walther, T., Menrad, A., Orzechowski, H.D., Siemeister, G., Paul, M., Schirner, M. FASEB J. (2003) [Pubmed]
  2. Fibrosis rather than blood pressure determines cardiac BNP expression in mice. Walther, T., Klostermann, K., Heringer-Walther, S., Schultheiss, H.P., Tschöpe, C., Stepan, H. Regul. Pept. (2003) [Pubmed]
  3. Ischemic injury in experimental stroke depends on angiotensin II. Walther, T., Olah, L., Harms, C., Maul, B., Bader, M., Hörtnagl, H., Schultheiss, H.P., Mies, G. FASEB J. (2002) [Pubmed]
  4. Fetal, neonatal cord, and maternal plasma concentrations of angiotensin-converting enzyme (ACE). Walther, T., Faber, R., Maul, B., Schultheiss, H.P., Siems, W.E., Stepan, H. Prenat. Diagn. (2002) [Pubmed]
  5. Natriuretic peptide system in fetal heart and circulation. Walther, T., Schultheiss, H.P., Tschöpe, C., Stepan, H. J. Hypertens. (2002) [Pubmed]
  6. AT1 receptor blockade increases cardiac bradykinin via neutral endopeptidase after induction of myocardial infarction in rats. Walther, T., Siems, W.E., Hauke, D., Spillmann, F., Dendorfer, A., Krause, W., Schultheiss, H.P., Tschöpe, C. FASEB J. (2002) [Pubmed]
 
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