Bruce F. Bebo
Neurological Sciences Institute
Oregon Health & Science University
Portland
USA
Name/email consistency: high
- Evidence that pregnancy specific glycoproteins regulate T-Cell function and inflammatory autoimmune disease during pregnancy. Bebo, B.F., Dveksler, G.S. Curr. Drug. Targets. Inflamm. Allergy (2005)
- Low-dose estrogen therapy ameliorates experimental autoimmune encephalomyelitis in two different inbred mouse strains. Bebo, B.F., Fyfe-Johnson, A., Adlard, K., Beam, A.G., Vandenbark, A.A., Offner, H. J. Immunol. (2001)
- Interleukin 7 is a potent co-stimulator of myelin specific T cells that enhances the adoptive transfer of experimental autoimmune encephalomyelitis. Bebo, B.F., Schuster, J.C., Adlard, K., Vandenbark, A.A., Offner, H. Cytokine (2000)
- Androgens alter the cytokine profile and reduce encephalitogenicity of myelin-reactive T cells. Bebo, B.F., Schuster, J.C., Vandenbark, A.A., Offner, H. J. Immunol. (1999)
- Gender differences in experimental autoimmune encephalomyelitis develop during the induction of the immune response to encephalitogenic peptides. Bebo, B.F., Schuster, J.C., Vandenbark, A.A., Offner, H. J. Neurosci. Res. (1998)
- Gonadal hormones influence the immune response to PLP 139-151 and the clinical course of relapsing experimental autoimmune encephalomyelitis. Bebo, B.F., Zelinka-Vincent, E., Adamus, G., Amundson, D., Vandenbark, A.A., Offner, H. J. Neuroimmunol. (1998)
- Hypothesis: a possible role for mast cells and their inflammatory mediators in the pathogenesis of autoimmune encephalomyelitis. Bebo, B.F., Yong, T., Orr, E.L., Linthicum, D.S. J. Neurosci. Res. (1996)
