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Hiroe Toba

Department of Clinical Pharmacology

Division of Pathological Sciences

Kyoto Pharmaceutical University

Kyoto

Japan

[email]@mb.kyoto-phu.ac.jp

Name/email consistency: high

 
 
 
 
 
 
 

Affiliation

  • Department of Clinical Pharmacology, Division of Pathological Sciences, Kyoto Pharmaceutical University, Kyoto, Japan. 2005 - 2012

References

  1. Erythropoietin attenuated vascular dysfunction and inflammation by inhibiting NADPH oxidase-derived superoxide production in nitric oxide synthase-inhibited hypertensive rat aorta. Toba, H., Kojima, Y., Wang, J., Noda, K., Tian, W., Kobara, M., Nakata, T. Eur. J. Pharmacol. (2012) [Pubmed]
  2. L/N-type calcium channel blocker cilnidipine ameliorates proteinuria and inhibits the renal renin-angiotensin-aldosterone system in deoxycorticosterone acetate-salt hypertensive rats. Toba, H., Yoshida, M., Tojo, C., Nakano, A., Oshima, Y., Kojima, Y., Noda, K., Wang, J., Kobara, M., Nakata, T. Hypertens. Res. (2011) [Pubmed]
  3. Recombinant human erythropoietin ameliorated endothelial dysfunction and macrophage infiltration by increasing nitric oxide in hypertensive 5/6 nephrectomized rat aorta. Toba, H., Morishita, M., Tojo, C., Nakano, A., Oshima, Y., Kojima, Y., Yoshida, M., Nakashima, K., Wang, J., Kobara, M., Nakata, T. Eur. J. Pharmacol. (2011) [Pubmed]
  4. Erythropoietin prevents vascular inflammation and oxidative stress in subtotal nephrectomized rat aorta beyond haematopoiesis. Toba, H., Nakashima, K., Oshima, Y., Kojima, Y., Tojo, C., Nakano, A., Wang, J., Kobara, M., Nakata, T. Clin. Exp. Pharmacol. Physiol. (2010) [Pubmed]
  5. Oral L-histidine exerts antihypertensive effects via central histamine H3 receptors and decreases nitric oxide content in the rostral ventrolateral medulla in spontaneously hypertensive rats. Toba, H., Nakamori, A., Tanaka, Y., Yukiya, R., Tatsuoka, K., Narutaki, M., Tokitaka, M., Hariu, H., Kobara, M., Nakata, T. Clin. Exp. Pharmacol. Physiol. (2010) [Pubmed]
  6. Inhibition of the renal renin-angiotensin system and renoprotection by pitavastatin in type1 diabetes. Toba, H., Mitani, T., Takahashi, T., Imai, N., Serizawa, R., Wang, J., Kobara, M., Nakata, T. Clin. Exp. Pharmacol. Physiol. (2010) [Pubmed]
  7. Chronic treatment with recombinant human erythropoietin exerts renoprotective effects beyond hematopoiesis in streptozotocin-induced diabetic rat. Toba, H., Sawai, N., Morishita, M., Murata, S., Yoshida, M., Nakashima, K., Morita, Y., Kobara, M., Nakata, T. Eur. J. Pharmacol. (2009) [Pubmed]
  8. Hyperinsulinaemia increases the gene expression of endothelial nitric oxide synthase and the phosphatidylinositol 3-kinase/Akt pathway in rat aorta. Toba, H., Gomyo, E., Miki, S., Shimizu, T., Yoshimura, A., Inoue, R., Sawai, N., Tsukamoto, R., Asayama, J., Kobara, M., Nakata, T. Clin. Exp. Pharmacol. Physiol. (2006) [Pubmed]
  9. Calcium [corrected] channel blockers reduce angiotensin II-induced superoxide generation and inhibit lectin-like oxidized low-density lipoprotein receptor-1 expression in endothelial cells. Toba, H., Shimizu, T., Miki, S., Inoue, R., Yoshimura, A., Tsukamoto, R., Sawai, N., Kobara, M., Nakata, T. Hypertens. Res. (2006) [Pubmed]
  10. The direct antioxidative and anti-inflammatory effects of peroxisome proliferator-activated receptors ligands are associated with the inhibition of angiotensin converting enzyme expression in streptozotocin-induced diabetic rat aorta. Toba, H., Miki, S., Shimizu, T., Yoshimura, A., Inoue, R., Sawai, N., Tsukamoto, R., Murakami, M., Morita, Y., Nakayama, Y., Kobara, M., Nakata, T. Eur. J. Pharmacol. (2006) [Pubmed]
  11. Calcium channel blockades exhibit anti-inflammatory and antioxidative effects by augmentation of endothelial nitric oxide synthase and the inhibition of angiotensin converting enzyme in the N(G)-nitro-L-arginine methyl ester-induced hypertensive rat aorta: vasoprotective effects beyond the blood pressure-lowering effects of amlodipine and manidipine. Toba, H., Nakagawa, Y., Miki, S., Shimizu, T., Yoshimura, A., Inoue, R., Asayama, J., Kobara, M., Nakata, T. Hypertens. Res. (2005) [Pubmed]
 
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