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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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V.M. Richon

DeWitt Wallace Research Laboratory

Memorial Sloan-Kettering Cancer Center

New York

New York 10021

USA

[email]@ski.mskcc.org

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • DeWitt Wallace Research Laboratory, Memorial Sloan-Kettering Cancer Center, New York, New York 10021, USA. 1996 - 2001
  • DeWitt Wallace Research Laboratory, Cell Biology Program, Memorial Sloan-Kettering Cancer Center and Graduate School of Medical Sciences of Cornell Medical School, New York, USA. 2000

References

  1. Histone deacetylase inhibitors: development of suberoylanilide hydroxamic acid (SAHA) for the treatment of cancers. Richon, V.M., Zhou, X., Rifkind, R.A., Marks, P.A. Blood Cells Mol. Dis. (2001) [Pubmed]
  2. Histone deacetylase inhibitor selectively induces p21WAF1 expression and gene-associated histone acetylation. Richon, V.M., Sandhoff, T.W., Rifkind, R.A., Marks, P.A. Proc. Natl. Acad. Sci. U.S.A. (2000) [Pubmed]
  3. A class of hybrid polar inducers of transformed cell differentiation inhibits histone deacetylases. Richon, V.M., Emiliani, S., Verdin, E., Webb, Y., Breslow, R., Rifkind, R.A., Marks, P.A. Proc. Natl. Acad. Sci. U.S.A. (1998) [Pubmed]
  4. Two cytodifferentiation agent-induced pathways, differentiation and apoptosis, are distinguished by the expression of human papillomavirus 16 E7 in human bladder carcinoma cells. Richon, V.M., Russo, P., Venta-Perez, G., Cordon-Cardo, C., Rifkind, R.A., Marks, P.A. Cancer Res. (1997) [Pubmed]
  5. Changes in E2F DNA-binding activity during induced erythroid differentiation. Richon, V.M., Venta-Perez, G. Cell Growth Differ. (1996) [Pubmed]
 
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