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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)

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Wendy B. Bollinger Bollag

Program in Cell Signaling

Department of Medicine

Institute of Molecular Medicine and Genetics

Medical College of Georgia

USA

[email]@mail.mcg.edu

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Affiliations

Program in Cell Signaling, Department of Medicine, Institute of Molecular Medicine and Genetics, Medical College of Georgia, USA. 2001 - 2005
Department of Medicine (Dermatology), Medical College of Georgia, Augusta, GA 30912 USA, USA. 2004

References

Phospholipase d signaling and extracellular signal-regulated kinase-1 and -2 phosphorylation (activation) are required for maximal phorbol ester-induced transglutaminase activity, a marker of keratinocyte differentiation. Bollag, W.B., Zhong, X., Dodd, M.E., Hardy, D.M., Zheng, X., Allred, W.T. J. Pharmacol. Exp. Ther. (2005) [Pubmed]
Protein kinase D and keratinocyte proliferation. Bollag, W.B., Dodd, M.E., Shapiro, B.A. Drug News Perspect. (2004) [Pubmed]
8-Cl-Adenosine enhances 1,25-dihydroxyvitamin D3-induced growth inhibition without affecting 1,25-dihydroxyvitamin D3-stimulated differentiation of primary mouse epidermal keratinocytes. Bollag, W.B., Zhong, X., Josephson, S. BMC Pharmacol. (2004) [Pubmed]
Mechanism of angiotensin II-induced phospholipase D activation in bovine adrenal glomerulosa cells. Bollag, W.B., Jung, E., Calle, R.A. Mol. Cell. Endocrinol. (2002) [Pubmed]
1,25-Dihydroxyvitamin D(3), phospholipase D and protein kinase C in keratinocyte differentiation. Bollinger Bollag, W., Bollag, R.J. Mol. Cell. Endocrinol. (2001) [Pubmed]

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