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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)

Conrad C. Weihl

Department of Neurology

Washington University School of Medicine

Saint Louis

MO 63110



Name/email consistency: high



  • Department of Neurology, Washington University School of Medicine, Saint Louis, MO 63110, USA. 2006 - 2011
  • Department of Neurology and Hope Center for Neurological Disorders, Washington University School of Medicine, 660 S. Euclid Avenue, Saint Louis, USA. 2011


  1. Valosin containing protein associated fronto-temporal lobar degeneration: clinical presentation, pathologic features and pathogenesis. Weihl, C.C. Curr. Alzheimer. Res (2011) [Pubmed]
  2. Novel GNE mutations in two phenotypically distinct HIBM2 patients. Weihl, C.C., Miller, S.E., Zaidman, C.M., Pestronk, A., Baloh, R.H., Al-Lozi, M. Neuromuscul. Disord. (2011) [Pubmed]
  3. Valosin-containing protein disease: inclusion body myopathy with Paget's disease of the bone and fronto-temporal dementia. Weihl, C.C., Pestronk, A., Kimonis, V.E. Neuromuscul. Disord. (2009) [Pubmed]
  4. TDP-43 accumulation in inclusion body myopathy muscle suggests a common pathogenic mechanism with frontotemporal dementia. Weihl, C.C., Temiz, P., Miller, S.E., Watts, G., Smith, C., Forman, M., Hanson, P.I., Kimonis, V., Pestronk, A. J. Neurol. Neurosurg. Psychiatr. (2008) [Pubmed]
  5. Transgenic expression of inclusion body myopathy associated mutant p97/VCP causes weakness and ubiquitinated protein inclusions in mice. Weihl, C.C., Miller, S.E., Hanson, P.I., Pestronk, A. Hum. Mol. Genet. (2007) [Pubmed]
  6. Inclusion body myopathy-associated mutations in p97/VCP impair endoplasmic reticulum-associated degradation. Weihl, C.C., Dalal, S., Pestronk, A., Hanson, P.I. Hum. Mol. Genet. (2006) [Pubmed]
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