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Walmor C. De Mello

Department of Pharmacology

School of Medicine Medical Sciences Campus

UPR, San Juan

PR 00936-5067

USA

[email]@rcm.upr.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Department of Pharmacology, School of Medicine Medical Sciences Campus, UPR, San Juan, PR 00936-5067, USA. 2003 - 2009
  • Medical Sciences Campus, UPR, School of Medicine, San Juan. 2008
  • Department of Pharmacology, UPR, San Juan, Puerto Rico 00936-5067, USA. 2001 - 2006

References

  1. Opposite effects of angiotensin II and angiotensin (1-7) on impulse propagation, excitability and cardiac arrhythmias. Is the overexpression of ACE2 arrhythmogenic? De Mello, W.C. Regul. Pept. (2009) [Pubmed]
  2. Intracellular and extracellular renin have opposite effects on the regulation of heart cell volume. Implications for myocardial ischaemia. De Mello, W.C. J. Renin. Angiotensin. Aldosterone. Syst (2008) [Pubmed]
  3. Eplerenone inhibits the intracrine and extracellular actions of angiotensin II on the inward calcium current in the failing heart. On the presence of an intracrine renin angiotensin aldosterone system. De Mello, W.C., Gerena, Y. Regul. Pept. (2008) [Pubmed]
  4. Beneficial versus harmful effects of Angiotensin (1-7) on impulse propagation and cardiac arrhythmias in the failing heart. De Mello, W.C., Ferrario, C.M., Jessup, J.A. J. Renin. Angiotensin. Aldosterone. Syst (2007) [Pubmed]
  5. Impaired cell communication in the diabetic heart. The role of the renin angiotensin system. De Mello, W.C. Mol. Cell. Biochem. (2007) [Pubmed]
  6. Chronic blockade of angiotensin II AT1-receptors increased cell-to-cell communication, reduced fibrosis and improved impulse propagation in the failing heart. De Mello, W.C., Specht, P. J. Renin. Angiotensin. Aldosterone. Syst (2006) [Pubmed]
  7. Beneficial effect of eplerenone on cardiac remodelling and electrical properties of the failing heart. De Mello, W.C. J. Renin. Angiotensin. Aldosterone. Syst (2006) [Pubmed]
  8. Cardiac intracrine renin angiotensin system. Part of genetic reprogramming?. De Mello, W.C. Regul. Pept. (2006) [Pubmed]
  9. Renin increments the inward calcium current in the failing heart. De Mello, W.C. J. Hypertens. (2006) [Pubmed]
  10. Angiotensin converting enzyme and the arrhythmogenic action of angiotensin I: cardiac cell membrane as a site of angiotensin I conversion. De Mello, W.C. Regul. Pept. (2004) [Pubmed]
  11. Heart failure: how important is cellular sequestration? The role of the renin-angiotensin-aldosterone system. De Mello, W.C. J. Mol. Cell. Cardiol. (2004) [Pubmed]
  12. Intracellular and extracellular angiotensin II enhance the L-type calcium current in the failing heart. De Mello, W.C., Monterrubio, J. Hypertension (2004) [Pubmed]
  13. Angiotensin (1-7) re-establishes impulse conduction in cardiac muscle during ischaemia-reperfusion. The role of the sodium pump. De Mello, W.C. J. Renin. Angiotensin. Aldosterone. Syst (2004) [Pubmed]
  14. Effect of extracellular and intracellular angiotensins on heart cell function; on the cardiac renin-angiotensin system. De Mello, W. Regul. Pept. (2003) [Pubmed]
  15. Further studies on the effect of intracellular angiotensins on heart cell communication: on the role of endogenous angiotensin II. De Mello, W.C. Regul. Pept. (2003) [Pubmed]
  16. Electrical activity of the heart and angiotensin-converting enzyme inhibitors on the hyperpolarising action of enalapril. De Mello, W.C. J. Hum. Hypertens (2002) [Pubmed]
  17. Cardiac arrhythmias: the possible role of the renin-angiotensin system. De Mello, W.C. J. Mol. Med. (2001) [Pubmed]
 
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