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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

Ye-Shih Ho

Institute of Environmental Health Sciences and Department of Biochemistry and Molecular Biology

Wayne State University

Detroit

MI 48201

USA

[email]@wayne.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Institute of Environmental Health Sciences and Department of Biochemistry and Molecular Biology, Wayne State University, Detroit, MI 48201, USA. 2002 - 2007
  • Institute of Chemical Toxicology, Wayne State University, Detroit, MI 48201, USA. 1998

References

  1. Targeted disruption of the glutaredoxin 1 gene does not sensitize adult mice to tissue injury induced by ischemia/reperfusion and hyperoxia. Ho, Y.S., Xiong, Y., Ho, D.S., Gao, J., Chua, B.H., Pai, H., Mieyal, J.J. Free Radic. Biol. Med. (2007) [Pubmed]
  2. Mice lacking catalase develop normally but show differential sensitivity to oxidant tissue injury. Ho, Y.S., Xiong, Y., Ma, W., Spector, A., Ho, D.S. J. Biol. Chem. (2004) [Pubmed]
  3. Transgenic and knockout models for studying the role of lung antioxidant enzymes in defense against hyperoxia. Ho, Y.S. Am. J. Respir. Crit. Care Med. (2002) [Pubmed]
  4. The nature of antioxidant defense mechanisms: a lesson from transgenic studies. Ho, Y.S., Magnenat, J.L., Gargano, M., Cao, J. Environ. Health Perspect. (1998) [Pubmed]
 
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