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Yuko Iwata

Department of Molecular Physiology

National Cardiovascular Center Research Institute Suita

Osaka 565-8565

Japan

[email]@ri.ncvc.go.jp

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Affiliation

Department of Molecular Physiology, National Cardiovascular Center Research Institute Suita, Osaka 565-8565, Japan. 2004 - 2009

References

Dominant-negative inhibition of Ca2+ influx via TRPV2 ameliorates muscular dystrophy in animal models. Iwata, Y., Katanosaka, Y., Arai, Y., Shigekawa, M., Wakabayashi, S. Hum. Mol. Genet. (2009) [Pubmed]
Cardiac syntrophin isoforms: species-dependent expression, association with dystrophin complex and subcellular localization. Iwata, Y., Shigekawa, M., Wakabayashi, S. Mol. Cell. Biochem. (2005) [Pubmed]
Protective effects of Ca2+ handling drugs against abnormal Ca2+ homeostasis and cell damage in myopathic skeletal muscle cells. Iwata, Y., Katanosaka, Y., Shijun, Z., Kobayashi, Y., Hanada, H., Shigekawa, M., Wakabayashi, S. Biochem. Pharmacol. (2005) [Pubmed]
Syntrophin is an actin-binding protein the cellular localization of which is regulated through cytoskeletal reorganization in skeletal muscle cells. Iwata, Y., Sampaolesi, M., Shigekawa, M., Wakabayashi, S. Eur. J. Cell Biol. (2004) [Pubmed]