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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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Bingbing Jiang

Whitaker Cardiovascular Institute

Vascular Biology Unit

Department of Medicine

Boston University School of Medicine

USA

[email]@bu.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliation

  • Whitaker Cardiovascular Institute, Vascular Biology Unit, Department of Medicine, Boston University School of Medicine, USA. 2001 - 2004

References

  1. Temporal control of NF-kappaB activation by ERK differentially regulates interleukin-1beta-induced gene expression. Jiang, B., Xu, S., Hou, X., Pimentel, D.R., Brecher, P., Cohen, R.A. J. Biol. Chem. (2004) [Pubmed]
  2. Angiotensin II differentially regulates interleukin-1-beta-inducible NO synthase (iNOS) and vascular cell adhesion molecule-1 (VCAM-1) expression: role of p38 MAPK. Jiang, B., Xu, S., Hou, X., Pimentel, D.R., Cohen, R.A. J. Biol. Chem. (2004) [Pubmed]
  3. Growth factors enhance interleukin-1 beta-induced persistent activation of nuclear factor-kappa B in rat vascular smooth muscle cells. Jiang, B., Xu, S., Brecher, P., Cohen, R.A. Arterioscler. Thromb. Vasc. Biol. (2002) [Pubmed]
  4. Persistent activation of nuclear factor-kappaB by interleukin-1beta and subsequent inducible NO synthase expression requires extracellular signal-regulated kinase. Jiang, B., Brecher, P., Cohen, R.A. Arterioscler. Thromb. Vasc. Biol. (2001) [Pubmed]
 
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