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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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Babu J. Padanilam

Dept. of Cellular and Integrative Physiology

Univ. of Nebraska Medical Center

Omaha

NE 68198-5850

[email]@unmc.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Dept. of Cellular and Integrative Physiology, Univ. of Nebraska Medical Center, Omaha, NE 68198-5850. 2003 - 2011
  • Renal Division, Department of Medicine, Washington University School of Medicine, St. Louis, USA. 2001

References

  1. Loss of poly(ADP-ribose) polymerase 1 attenuates renal fibrosis and inflammation during unilateral ureteral obstruction. Kim, J., Padanilam, B.J. Am. J. Physiol. Renal Physiol. (2011) [Pubmed]
  2. p53 target Siva regulates apoptosis in ischemic kidneys. Singaravelu, K., Padanilam, B.J. Am. J. Physiol. Renal Physiol. (2011) [Pubmed]
  3. Cell death induced by acute renal injury: a perspective on the contributions of apoptosis and necrosis. Padanilam, B.J. Am. J. Physiol. Renal Physiol. (2003) [Pubmed]
  4. Activation of protein kinase C isozymes protects LLCPK1 cells from H2O2 induced necrotic cell death. Polosukhina, D., Singaravelu, K., Padanilam, B.J. Am. J. Nephrol. (2003) [Pubmed]
  5. Induction and subcellular localization of protein kinase C isozymes following renal ischemia. Padanilam, B.J. Kidney Int. (2001) [Pubmed]
 
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