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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)

Brian C. Lewis

University of Massachusetts Medical School

364 Plantation Street

LRB 521



Name/email consistency: high



  • University of Massachusetts Medical School, 364 Plantation Street, LRB 521, Worcester. 2005 - 2012
  • Cancer Biology and Genetics Program, Memorial Sloan-Kettering Cancer Center, New York, NY 10021, USA. 2003


  1. KRASG12D- and BRAFV600E-Induced Transformation of Murine Pancreatic Epithelial Cells Requires MEK/ERK-Stimulated IGF1R Signaling. Appleman, V.A., Ahronian, L.G., Cai, J., Klimstra, D.S., Lewis, B.C. Mol. Cancer Res. (2012) [Pubmed]
  2. Trp53 deletion stimulates the formation of metastatic pancreatic tumors. Morton, J.P., Klimstra, D.S., Mongeau, M.E., Lewis, B.C. Am. J. Pathol. (2008) [Pubmed]
  3. Development of the pancreas and pancreatic cancer. Lewis, B.C. Endocrinol. Metab. Clin. North Am. (2006) [Pubmed]
  4. The absence of p53 promotes metastasis in a novel somatic mouse model for hepatocellular carcinoma. Lewis, B.C., Klimstra, D.S., Socci, N.D., Xu, S., Koutcher, J.A., Varmus, H.E. Mol. Cell. Biol. (2005) [Pubmed]
  5. The c-myc and PyMT oncogenes induce different tumor types in a somatic mouse model for pancreatic cancer. Lewis, B.C., Klimstra, D.S., Varmus, H.E. Genes Dev. (2003) [Pubmed]
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