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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

Dmitry V. Bulavin

Gene Response Section

National Cancer Institute

National Institutes of Health

Bethesda

USA

[email]@nih.gov

Name/email consistency: high

 
 
 
 
 
 
 

Affiliation

  • Gene Response Section, National Cancer Institute, National Institutes of Health, Bethesda, USA. 2003 - 2004

References

  1. Inactivation of the Wip1 phosphatase inhibits mammary tumorigenesis through p38 MAPK-mediated activation of the p16(Ink4a)-p19(Arf) pathway. Bulavin, D.V., Phillips, C., Nannenga, B., Timofeev, O., Donehower, L.A., Anderson, C.W., Appella, E., Fornace, A.J. Nat. Genet. (2004) [Pubmed]
  2. Phosphorylation of Xenopus Cdc25C at Ser285 interferes with ability to activate a DNA damage replication checkpoint in pre-midblastula embryos. Bulavin, D.V., Demidenko, Z.N., Phillips, C., Moody, S.A., Fornace, A.J. Cell. Cycle (2003) [Pubmed]
 
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