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Joseph D. Buxbaum

Laboratory of Molecular Neuropsychiatry

Department of Psychiatry

Mount Sinai School of Medicine

One Gustave L. Levy Place

USA

[email]@doc.mssm.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Laboratory of Molecular Neuropsychiatry, Department of Psychiatry, Mount Sinai School of Medicine, One Gustave L. Levy Place, USA. 2002
  • Department of Psychiatry, Mount Sinai School of Medicine, New York, New York 10029, USA. 1998 - 2001

References

  1. Pharmacological concentrations of the HMG-CoA reductase inhibitor lovastatin decrease the formation of the Alzheimer beta-amyloid peptide in vitro and in patients. Buxbaum, J.D., Cullen, E.I., Friedhoff, L.T. Front. Biosci. (2002) [Pubmed]
  2. Evidence for a susceptibility gene for autism on chromosome 2 and for genetic heterogeneity. Buxbaum, J.D., Silverman, J.M., Smith, C.J., Kilifarski, M., Reichert, J., Hollander, E., Lawlor, B.A., Fitzgerald, M., Greenberg, D.A., Davis, K.L. Am. J. Hum. Genet. (2001) [Pubmed]
  3. Genomic structure, expression pattern, and chromosomal localization of the human calsenilin gene: no association between an exonic polymorphism and Alzheimer's disease. Buxbaum, J.D., Lilliehook, C., Chan, J.Y., Go, R.C., Bassett, S.S., Tanzi, R.E., Wasco, W., Blacker, D. Neurosci. Lett. (2000) [Pubmed]
  4. Calsenilin: a calcium-binding protein that interacts with the presenilins and regulates the levels of a presenilin fragment. Buxbaum, J.D., Choi, E.K., Luo, Y., Lilliehook, C., Crowley, A.C., Merriam, D.E., Wasco, W. Nat. Med. (1998) [Pubmed]
  5. Evidence that tumor necrosis factor alpha converting enzyme is involved in regulated alpha-secretase cleavage of the Alzheimer amyloid protein precursor. Buxbaum, J.D., Liu, K.N., Luo, Y., Slack, J.L., Stocking, K.L., Peschon, J.J., Johnson, R.S., Castner, B.J., Cerretti, D.P., Black, R.A. J. Biol. Chem. (1998) [Pubmed]
 
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