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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

Barbara D. Abbott

Reproductive Toxicology Division

National Health and Environmental Effects Research Laboratory

Office of Research and Development

US Environmental Protection Agency

USA

[email]@epa.gov

Name/email consistency: high

 
 
 
 
 
 
 

Affiliation

  • Reproductive Toxicology Division, National Health and Environmental Effects Research Laboratory, Office of Research and Development, US Environmental Protection Agency, USA. 2000 - 2009

References

  1. Developmental toxicity of perfluorooctane sulfonate (PFOS) is not dependent on expression of peroxisome proliferator activated receptor-alpha (PPAR alpha) in the mouse. Abbott, B.D., Wolf, C.J., Das, K.P., Zehr, R.D., Schmid, J.E., Lindstrom, A.B., Strynar, M.J., Lau, C. Reprod. Toxicol. (2009) [Pubmed]
  2. Review of the expression of peroxisome proliferator-activated receptors alpha (PPAR alpha), beta (PPAR beta), and gamma (PPAR gamma) in rodent and human development. Abbott, B.D. Reprod. Toxicol. (2009) [Pubmed]
  3. Perfluorooctanoic acid induced developmental toxicity in the mouse is dependent on expression of peroxisome proliferator activated receptor-alpha. Abbott, B.D., Wolf, C.J., Schmid, J.E., Das, K.P., Zehr, R.D., Helfant, L., Nakayama, S., Lindstrom, A.B., Strynar, M.J., Lau, C. Toxicol. Sci. (2007) [Pubmed]
  4. Teratogenic effects of retinoic acid are modulated in mice lacking expression of epidermal growth factor and transforming growth factor-alpha. Abbott, B.D., Best, D.S., Narotsky, M.G. Birth Defects Res. Part A Clin. Mol. Teratol. (2005) [Pubmed]
  5. Effects of epidermal growth factor (EGF), transforming growth factor-alpha (TGFalpha), and 2,3,7,8-tetrachlorodibenzo-p-dioxin on fusion of embryonic palates in serum-free organ culture using wild-type, EGF knockout, and TGFalpha knockout mouse strains. Abbott, B.D., Buckalew, A.R., Leffler, K.E. Birth Defects Res. Part A Clin. Mol. Teratol. (2005) [Pubmed]
  6. EGF and TGF-alpha expression influence the developmental toxicity of TCDD: dose response and AhR phenotype in EGF, TGF-alpha, and EGF + TGF-alpha knockout mice. Abbott, B.D., Buckalew, A.R., DeVito, M.J., Ross, D., Bryant, P.L., Schmid, J.E. Toxicol. Sci. (2003) [Pubmed]
  7. Lack of expression of EGF and TGF-alpha in the fetal mouse alters formation of prostatic epithelial buds and influences the response to TCDD. Abbott, B.D., Lin, T.M., Rasmussen, N.T., Albrecht, R.M., Schmid, J.E., Peterson, R.E. Toxicol. Sci. (2003) [Pubmed]
  8. Placental defects in ARNT-knockout conceptus correlate with localized decreases in VEGF-R2, Ang-1, and Tie-2. Abbott, B.D., Buckalew, A.R. Dev. Dyn. (2000) [Pubmed]
 
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