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Paolo Calabresi

Dipartimento di Neuroscienze

Università di Roma Tor Vergata

Italy

[email]@uniroma2.it

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Dipartimento di Neuroscienze, Università di Roma Tor Vergata, Italy. 1998 - 2005
  • Clinica Neurologica, Dip. Neuroscienze, Università di Tor Vergata, Via di Tor Vergata 135, Italy. 2000 - 2003
  • Clinica Neurologica, Università Tor Vergata, Rome, 00133, Italy. 2000

References

  1. Medication-overuse headache: similarities with drug addiction. Calabresi, P., Cupini, L.M. Trends Pharmacol. Sci. (2005) [Pubmed]
  2. Ionotropic glutamate receptors: still a target for neuroprotection in brain ischemia? Insights from in vitro studies. Calabresi, P., Centonze, D., Cupini, L.M., Costa, C., Pisani, F., Bernardi, G. Neurobiol. Dis. (2003) [Pubmed]
  3. Antiepileptic drugs as a possible neuroprotective strategy in brain ischemia. Calabresi, P., Cupini, L.M., Centonze, D., Pisani, F., Bernardi, G. Ann. Neurol. (2003) [Pubmed]
  4. Lamotrigine and remacemide protect striatal neurons against in vitro ischemia: an electrophysiological study. Calabresi, P., Marti, M., Picconi, B., Saulle, E., Costa, C., Centonze, D., Pisani, F., Bernardi, G. Exp. Neurol. (2003) [Pubmed]
  5. Synaptic plasticity in the ischaemic brain. Calabresi, P., Centonze, D., Pisani, A., Cupini, L., Bernardi, G. Lancet. Neurol (2003) [Pubmed]
  6. Post-ischaemic long-term synaptic potentiation in the striatum: a putative mechanism for cell type-specific vulnerability. Calabresi, P., Saulle, E., Centonze, D., Pisani, A., Marfia, G.A., Bernardi, G. Brain (2002) [Pubmed]
  7. Activation of metabotropic glutamate receptor subtype 1/protein kinase C/mitogen-activated protein kinase pathway is required for postischemic long-term potentiation in the striatum. Calabresi, P., Saulle, E., Marfia, G.A., Centonze, D., Mulloy, R., Picconi, B., Hipskind, R.A., Conquet, F., Bernardi, G. Mol. Pharmacol. (2001) [Pubmed]
  8. Is pharmacological neuroprotection dependent on reduced glutamate release?. Calabresi, P., Picconi, B., Saulle, E., Centonze, D., Hainsworth, A.H., Bernardi, G. Stroke (2000) [Pubmed]
  9. Electrophysiology of sipatrigine: a lamotrigine derivative exhibiting neuroprotective effects. Calabresi, P., Stefani, A., Marfia, G.A., Hainsworth, A.H., Centonze, D., Saulle, E., Spadoni, F., Leach, M.J., Giacomini, P., Bernardi, G. Exp. Neurol. (2000) [Pubmed]
  10. Synaptic transmission in the striatum: from plasticity to neurodegeneration. Calabresi, P., Centonze, D., Gubellini, P., Marfia, G.A., Pisani, A., Sancesario, G., Bernardi, G. Prog. Neurobiol. (2000) [Pubmed]
  11. Tissue plasminogen activator controls multiple forms of synaptic plasticity and memory. Calabresi, P., Napolitano, M., Centonze, D., Marfia, G.A., Gubellini, P., Teule, M.A., Berretta, N., Bernardi, G., Frati, L., Tolu, M., Gulino, A. Eur. J. Neurosci. (2000) [Pubmed]
  12. Dopamine and cAMP-regulated phosphoprotein 32 kDa controls both striatal long-term depression and long-term potentiation, opposing forms of synaptic plasticity. Calabresi, P., Gubellini, P., Centonze, D., Picconi, B., Bernardi, G., Chergui, K., Svenningsson, P., Fienberg, A.A., Greengard, P. J. Neurosci. (2000) [Pubmed]
  13. Cellular factors controlling neuronal vulnerability in the brain: a lesson from the striatum. Calabresi, P., Centonze, D., Bernardi, G. Neurology (2000) [Pubmed]
  14. An in vitro electrophysiological study on the effects of phenytoin, lamotrigine and gabapentin on striatal neurons. Calabresi, P., Centonze, D., Marfia, G.A., Pisani, A., Bernardi, G. Br. J. Pharmacol. (1999) [Pubmed]
  15. Pharmacological inhibition of the Na(+)/Ca(2+) exchanger enhances depolarizations induced by oxygen/glucose deprivation but not responses to excitatory amino acids in rat striatal neurons. Calabresi, P., Marfia, G.A., Amoroso, S., Pisani, A., Bernardi, G. Stroke (1999) [Pubmed]
  16. Muscarinic IPSPs in rat striatal cholinergic interneurones. Calabresi, P., Centonze, D., Pisani, A., Sancesario, G., North, R.A., Bernardi, G. J. Physiol. (Lond.) (1998) [Pubmed]
  17. Endogenous ACh enhances striatal NMDA-responses via M1-like muscarinic receptors and PKC activation. Calabresi, P., Centonze, D., Gubellini, P., Pisani, A., Bernardi, G. Eur. J. Neurosci. (1998) [Pubmed]
 
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