C.D. Jun
Department of Microbiology and Immunology
Wonkwang University School of Medicine
Iksan
Chonbuk
Korea
Name/email consistency: high
- Overexpression of protein kinase C isoforms protects RAW 264.7 macrophages from nitric oxide-induced apoptosis: involvement of c-Jun N-terminal kinase/stress-activated protein kinase, p38 kinase, and CPP-32 protease pathways. Jun, C.D., Oh, C.D., Kwak, H.J., Pae, H.O., Yoo, J.C., Choi, B.M., Chun, J.S., Park, R.K., Chung, H.T. J. Immunol. (1999)
- Modulation of nitric oxide-induced apoptotic death of HL-60 cells by protein kinase C and protein kinase A through mitogen-activated protein kinases and CPP32-like protease pathways. Jun, C.D., Pae, H.O., Kwak, H.J., Yoo, J.C., Choi, B.M., Oh, C.D., Chun, J.S., Paik, S.G., Park, Y.H., Chung, H.T. Cell. Immunol. (1999)