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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

Carl G. Feng

Laboratory of Parasitic Diseases

National Institute of Allergy and Infectious Diseases

National Institutes of Health

Bethesda

USA

[email]@niaid.nih.gov

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Laboratory of Parasitic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, USA. 2002 - 2009
  • Immunobiology Section, National Institute of Allergy and Infectious Diseases, Bethesda, MD 20892-8003, USA. 2006

References

  1. Interferon-inducible immunity-related GTPase Irgm1 regulates IFN gamma-dependent host defense, lymphocyte survival and autophagy. Feng, C.G., Zheng, L., Lenardo, M.J., Sher, A. Autophagy (2009) [Pubmed]
  2. The immunity-related GTPase Irgm1 promotes the expansion of activated CD4+ T cell populations by preventing interferon-gamma-induced cell death. Feng, C.G., Zheng, L., Jankovic, D., Báfica, A., Cannons, J.L., Watford, W.T., Chaussabel, D., Hieny, S., Caspar, P., Schwartzberg, P.L., Lenardo, M.J., Sher, A. Nat. Immunol. (2008) [Pubmed]
  3. NK cell-derived IFN-gamma differentially regulates innate resistance and neutrophil response in T cell-deficient hosts infected with Mycobacterium tuberculosis. Feng, C.G., Kaviratne, M., Rothfuchs, A.G., Cheever, A., Hieny, S., Young, H.A., Wynn, T.A., Sher, A. J. Immunol. (2006) [Pubmed]
  4. Maintenance of pulmonary Th1 effector function in chronic tuberculosis requires persistent IL-12 production. Feng, C.G., Jankovic, D., Kullberg, M., Cheever, A., Scanga, C.A., Hieny, S., Caspar, P., Yap, G.S., Sher, A. J. Immunol. (2005) [Pubmed]
  5. Mice deficient in LRG-47 display increased susceptibility to mycobacterial infection associated with the induction of lymphopenia. Feng, C.G., Collazo-Custodio, C.M., Eckhaus, M., Hieny, S., Belkaid, Y., Elkins, K., Jankovic, D., Taylor, G.A., Sher, A. J. Immunol. (2004) [Pubmed]
  6. Mice lacking myeloid differentiation factor 88 display profound defects in host resistance and immune responses to Mycobacterium avium infection not exhibited by Toll-like receptor 2 (TLR2)- and TLR4-deficient animals. Feng, C.G., Scanga, C.A., Collazo-Custodio, C.M., Cheever, A.W., Hieny, S., Caspar, P., Sher, A. J. Immunol. (2003) [Pubmed]
  7. Transgenic mice expressing human interleukin-10 in the antigen-presenting cell compartment show increased susceptibility to infection with Mycobacterium avium associated with decreased macrophage effector function and apoptosis. Feng, C.G., Kullberg, M.C., Jankovic, D., Cheever, A.W., Caspar, P., Coffman, R.L., Sher, A. Infect. Immun. (2002) [Pubmed]
 
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