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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)

Jae Jin Chae

Laboratory of Clinical Investigation

National Institutes of Arthritis and Musculoskeletal and Skin Diseases

Genetics and Genomics Branch




Name/email consistency: high



  • Laboratory of Clinical Investigation, National Institutes of Arthritis and Musculoskeletal and Skin Diseases, Genetics and Genomics Branch, Bethesda, USA. 2003 - 2009


  1. Advances in the understanding of familial Mediterranean fever and possibilities for targeted therapy. Chae, J.J., Aksentijevich, I., Kastner, D.L. Br. J. Haematol. (2009) [Pubmed]
  2. The familial Mediterranean fever protein, pyrin, is cleaved by caspase-1 and activates NF-kappaB through its N-terminal fragment. Chae, J.J., Wood, G., Richard, K., Jaffe, H., Colburn, N.T., Masters, S.L., Gumucio, D.L., Shoham, N.G., Kastner, D.L. Blood (2008) [Pubmed]
  3. The B30.2 domain of pyrin, the familial Mediterranean fever protein, interacts directly with caspase-1 to modulate IL-1beta production. Chae, J.J., Wood, G., Masters, S.L., Richard, K., Park, G., Smith, B.J., Kastner, D.L. Proc. Natl. Acad. Sci. U.S.A. (2006) [Pubmed]
  4. Targeted disruption of pyrin, the FMF protein, causes heightened sensitivity to endotoxin and a defect in macrophage apoptosis. Chae, J.J., Komarow, H.D., Cheng, J., Wood, G., Raben, N., Liu, P.P., Kastner, D.L. Mol. Cell (2003) [Pubmed]
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