Bysani Chandrasekar
Department of Veterans Affairs South Texas Veterans Health Care System
San Antonio
Texas 78229-4404
USA
Name/email consistency: high
- Interleukin-18 suppresses adiponectin expression in 3T3-L1 adipocytes via a novel signal transduction pathway involving ERK1/2-dependent NFATc4 phosphorylation. Chandrasekar, B., Patel, D.N., Mummidi, S., Kim, J.W., Clark, R.A., Valente, A.J. J. Biol. Chem. (2008)
- Adiponectin blocks interleukin-18-mediated endothelial cell death via APPL1-dependent AMP-activated protein kinase (AMPK) activation and IKK/NF-kappaB/PTEN suppression. Chandrasekar, B., Boylston, W.H., Venkatachalam, K., Webster, N.J., Prabhu, S.D., Valente, A.J. J. Biol. Chem. (2008)
- Interleukin-18 induces human cardiac endothelial cell death via a novel signaling pathway involving NF-kappaB-dependent PTEN activation. Chandrasekar, B., Valente, A.J., Freeman, G.L., Mahimainathan, L., Mummidi, S. Biochem. Biophys. Res. Commun. (2006)
- Interleukin-18-induced human coronary artery smooth muscle cell migration is dependent on NF-kappaB- and AP-1-mediated matrix metalloproteinase-9 expression and is inhibited by atorvastatin. Chandrasekar, B., Mummidi, S., Mahimainathan, L., Patel, D.N., Bailey, S.R., Imam, S.Z., Greene, W.C., Valente, A.J. J. Biol. Chem. (2006)
- The pro-atherogenic cytokine interleukin-18 induces CXCL16 expression in rat aortic smooth muscle cells via MyD88, interleukin-1 receptor-associated kinase, tumor necrosis factor receptor-associated factor 6, c-Src, phosphatidylinositol 3-kinase, Akt, c-Jun N-terminal kinase, and activator protein-1 signaling. Chandrasekar, B., Mummidi, S., Valente, A.J., Patel, D.N., Bailey, S.R., Freeman, G.L., Hatano, M., Tokuhisa, T., Jensen, L.E. J. Biol. Chem. (2005)
- CXCL16 signals via Gi, phosphatidylinositol 3-kinase, Akt, I kappa B kinase, and nuclear factor-kappa B and induces cell-cell adhesion and aortic smooth muscle cell proliferation. Chandrasekar, B., Bysani, S., Mummidi, S. J. Biol. Chem. (2004)
- Activation of intrinsic and extrinsic proapoptotic signaling pathways in interleukin-18-mediated human cardiac endothelial cell death. Chandrasekar, B., Vemula, K., Surabhi, R.M., Li-Weber, M., Owen-Schaub, L.B., Jensen, L.E., Mummidi, S. J. Biol. Chem. (2004)
- Beta-adrenergic stimulation induces interleukin-18 expression via beta2-AR, PI3K, Akt, IKK, and NF-kappaB. Chandrasekar, B., Marelli-Berg, F.M., Tone, M., Bysani, S., Prabhu, S.D., Murray, D.R. Biochem. Biophys. Res. Commun. (2004)
- Chemokine-cytokine cross-talk. The ELR+ CXC chemokine LIX (CXCL5) amplifies a proinflammatory cytokine response via a phosphatidylinositol 3-kinase-NF-kappa B pathway. Chandrasekar, B., Melby, P.C., Sarau, H.M., Raveendran, M., Perla, R.P., Marelli-Berg, F.M., Dulin, N.O., Singh, I.S. J. Biol. Chem. (2003)
- TNF-alpha and H2O2 induce IL-18 and IL-18R beta expression in cardiomyocytes via NF-kappa B activation. Chandrasekar, B., Colston, J.T., de la Rosa, S.D., Rao, P.P., Freeman, G.L. Biochem. Biophys. Res. Commun. (2003)
- Fractalkine (CX3CL1) stimulated by nuclear factor kappaB (NF-kappaB)-dependent inflammatory signals induces aortic smooth muscle cell proliferation through an autocrine pathway. Chandrasekar, B., Mummidi, S., Perla, R.P., Bysani, S., Dulin, N.O., Liu, F., Melby, P.C. Biochem. J. (2003)
- Calorie restriction attenuates inflammatory responses to myocardial ischemia-reperfusion injury. Chandrasekar, B., Nelson, J.F., Colston, J.T., Freeman, G.L. Am. J. Physiol. Heart Circ. Physiol. (2001)
- Ischemia-reperfusion of rat myocardium activates nuclear factor-KappaB and induces neutrophil infiltration via lipopolysaccharide-induced CXC chemokine. Chandrasekar, B., Smith, J.B., Freeman, G.L. Circulation (2001)
