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Claire H. Seedhouse

Division of Haematology

University of Nottingham

Nottingham

UK

[email]@nottingham.ac.uk

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Division of Haematology, University of Nottingham, Nottingham, UK. 2009 - 2012
  • Academic Haematology, Nottingham University Hospitals, University of Nottingham, Nottingham, United Kingdom. 2006 - 2007
  • Division of Haematology, School of Clinical Laboratory Sciences, University of Nottingham, and Nottingham City Hospital, United Kingdom. 2002 - 2004

References

  1. Targeting of CD34+CD38- cells using Gemtuzumab ozogamicin (Mylotarg) in combination with tipifarnib (Zarnestra) in acute Myeloid Leukaemia. Jawad, M., Yu, N., Seedhouse, C., Tandon, K., Russell, N.H., Pallis, M. BMC. Cancer (2012) [Pubmed]
  2. FLT3-ITD expression levels and their effect on STAT5 in AML with and without NPM mutations. Seedhouse, C.H., Pallis, M., Grundy, M., Shang, S., Russell, N.H. Br. J. Haematol. (2009) [Pubmed]
  3. Impaired S-phase arrest in acute myeloid leukemia cells with a FLT3 internal tandem duplication treated with clofarabine. Seedhouse, C., Grundy, M., Shang, S., Ronan, J., Pimblett, H., Russell, N., Pallis, M. Clin. Cancer Res. (2009) [Pubmed]
  4. Sequential influences of leukemia-specific and genetic factors on p-glycoprotein expression in blasts from 817 patients entered into the National Cancer Research Network acute myeloid leukemia 14 and 15 trials. Seedhouse, C.H., Grundy, M., White, P., Li, Y., Fisher, J., Yakunina, D., Moorman, A.V., Hoy, T., Russell, N., Burnett, A., Pallis, M. Clin. Cancer Res. (2007) [Pubmed]
  5. Advances in the understanding of susceptibility to treatment-related acute myeloid leukaemia. Seedhouse, C., Russell, N. Br. J. Haematol. (2007) [Pubmed]
  6. DNA repair contributes to the drug-resistant phenotype of primary acute myeloid leukaemia cells with FLT3 internal tandem duplications and is reversed by the FLT3 inhibitor PKC412. Seedhouse, C.H., Hunter, H.M., Lloyd-Lewis, B., Massip, A.M., Pallis, M., Carter, G.I., Grundy, M., Shang, S., Russell, N.H. Leukemia (2006) [Pubmed]
  7. Polymorphisms in genes involved in homologous recombination repair interact to increase the risk of developing acute myeloid leukemia. Seedhouse, C., Faulkner, R., Ashraf, N., Das-Gupta, E., Russell, N. Clin. Cancer Res. (2004) [Pubmed]
  8. The genotype distribution of the XRCC1 gene indicates a role for base excision repair in the development of therapy-related acute myeloblastic leukemia. Seedhouse, C., Bainton, R., Lewis, M., Harding, A., Russell, N., Das-Gupta, E. Blood (2002) [Pubmed]
 
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