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Agata Copani

Department of Pharmaceutical Sciences

University of Catania

95125 Catania

Italy

[email]@katamail.com

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Department of Pharmaceutical Sciences, University of Catania, 95125 Catania, Italy. 2001 - 2008
  • Department of Chemical Sciences, Section of Biochemistry and Molecular Biology, University of Catania, Italy. 2002

References

  1. The cell cycle molecules behind neurodegeneration in Alzheimer's disease: perspectives for drug development. Copani, A., Guccione, S., Giurato, L., Caraci, F., Calafiore, M., Sortino, M.A., Nicoletti, F. Curr. Med. Chem. (2008) [Pubmed]
  2. DNA polymerase-beta is expressed early in neurons of Alzheimer's disease brain and is loaded into DNA replication forks in neurons challenged with beta-amyloid. Copani, A., Hoozemans, J.J., Caraci, F., Calafiore, M., Van Haastert, E.S., Veerhuis, R., Rozemuller, A.J., Aronica, E., Sortino, M.A., Nicoletti, F. J. Neurosci. (2006) [Pubmed]
  3. Alzheimer's disease research enters a "new cycle": how significant?. Copani, A., Sortino, M.A., Nicoletti, F., Giuffrida, S.A. Neurochem. Res. (2002) [Pubmed]
  4. Beta-amyloid-induced synthesis of the ganglioside GD3 is a requisite for cell cycle reactivation and apoptosis in neurons. Copani, A., Melchiorri, D., Caricasole, A., Martini, F., Sale, P., Carnevale, R., Gradini, R., Sortino, M.A., Lenti, L., De Maria, R., Nicoletti, F. J. Neurosci. (2002) [Pubmed]
  5. Erratic expression of DNA polymerases by beta-amyloid causes neuronal death. Copani, A., Sortino, M.A., Caricasole, A., Chiechio, S., Chisari, M., Battaglia, G., Giuffrida-Stella, A.M., Vancheri, C., Nicoletti, F. FASEB J. (2002) [Pubmed]
  6. Cell cycle progression towards Alzheimer's disease. Copani, A., Condorelli, F., Canonico, P.L., Nicoletti, F., Sortino, M.A. Funct. Neurol. (2001) [Pubmed]
 
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