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Craig F. Ferris

Program in Behavioral Neuroscience

Department of Psychiatry

University of Massachusetts Medical School

Worcester 01655

USA

[email]@umassmed.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Program in Behavioral Neuroscience, Department of Psychiatry, University of Massachusetts Medical School, Worcester 01655, USA. 2001 - 2006
  • Center for Comparative Neuroimaging, University of Massachusetts Medical School, 55 Lake Avenue North, Worcester, USA. 2003 - 2006

References

  1. Orally active vasopressin V1a receptor antagonist, SRX251, selectively blocks aggressive behavior. Ferris, C.F., Lu, S.F., Messenger, T., Guillon, C.D., Heindel, N., Miller, M., Koppel, G., Robert Bruns, F., Simon, N.G. Pharmacol. Biochem. Behav. (2006) [Pubmed]
  2. Functional magnetic resonance imaging in conscious animals: a new tool in behavioural neuroscience research. Ferris, C.F., Febo, M., Luo, F., Schmidt, K., Brevard, M., Harder, J.A., Kulkarni, P., Messenger, T., King, J.A. J. Neuroendocrinol. (2006) [Pubmed]
  3. Pup suckling is more rewarding than cocaine: evidence from functional magnetic resonance imaging and three-dimensional computational analysis. Ferris, C.F., Kulkarni, P., Sullivan, J.M., Harder, J.A., Messenger, T.L., Febo, M. J. Neurosci. (2005) [Pubmed]
  4. Behavioral and neuroendocrine consequences of social subjugation across adolescence and adulthood. Ferris, C.F., Messenger, T., Sullivan, R. Front. Zool. (2005) [Pubmed]
  5. Activation of neural pathways associated with sexual arousal in non-human primates. Ferris, C.F., Snowdon, C.T., King, J.A., Sullivan, J.M., Ziegler, T.E., Olson, D.P., Schultz-Darken, N.J., Tannenbaum, P.L., Ludwig, R., Wu, Z., Einspanier, A., Vaughan, J.T., Duong, T.Q. J. Magn. Reson. Imaging (2004) [Pubmed]
  6. Using an animal model to assess the long-term behavioral and biological consequences of adolescent abuse and exposure to alcohol. Ferris, C.F. Ann. N. Y. Acad. Sci. (2003) [Pubmed]
  7. Vasopressin-dependent flank marking in golden hamsters is suppressed by drugs used in the treatment of obsessive-compulsive disorder. Ferris, C.F., Rasmussen, M.F., Messenger, T., Koppel, G. BMC. Neurosci (2001) [Pubmed]
 
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