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Corrine K. Welt

Reproductive Endocrine

BHX 511

Massachusetts General Hospital

55 Fruit Street

[email]@partners.org

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Reproductive Endocrine, BHX 511, Massachusetts General Hospital, 55 Fruit Street. 2003 - 2012
  • Reproductive Endocrine Unit, Reproductive Endocrine Sciences Center, National Center for Infertility Research, Department of Medicine, USA. 2001 - 2003

References

  1. Variants in DENND1A Are Associated with Polycystic Ovary Syndrome in Women of European Ancestry. Welt, C.K., Styrkarsdottir, U., Ehrmann, D.A., Thorleifsson, G., Arason, G., Gudmundsson, J.A., Ober, C., Rosenfield, R.L., Saxena, R., Thorsteinsdottir, U., Crowley, W.F., Stefansson, K. J. Clin. Endocrinol. Metab. (2012) [Pubmed]
  2. Autoimmune oophoritis in the adolescent. Welt, C.K. Ann. N. Y. Acad. Sci. (2008) [Pubmed]
  3. Primary ovarian insufficiency: a more accurate term for premature ovarian failure. Welt, C.K. Clin. Endocrinol. (Oxf) (2008) [Pubmed]
  4. Will leptin become the treatment of choice for functional hypothalamic amenorrhea?. Welt, C.K. Nature Clinical Practice. Endocrinology & Metabolism (2007) [Pubmed]
  5. Control of estradiol secretion in reproductive ageing. Welt, C.K., Jimenez, Y., Sluss, P.M., Smith, P.C., Hall, J.E. Hum. Reprod. (2006) [Pubmed]
  6. Defining constant versus variable phenotypic features of women with polycystic ovary syndrome using different ethnic groups and populations. Welt, C.K., Arason, G., Gudmundsson, J.A., Adams, J., Palsdóttir, H., Gudlaugsdóttir, G., Ingadóttir, G., Crowley, W.F. J. Clin. Endocrinol. Metab. (2006) [Pubmed]
  7. Characterizing discrete subsets of polycystic ovary syndrome as defined by the Rotterdam criteria: the impact of weight on phenotype and metabolic features. Welt, C.K., Gudmundsson, J.A., Arason, G., Adams, J., Palsdottir, H., Gudlaugsdottir, G., Ingadottir, G., Crowley, W.F. J. Clin. Endocrinol. Metab. (2006) [Pubmed]
  8. Relationship of estradiol and inhibin to the follicle-stimulating hormone variability in hypergonadotropic hypogonadism or premature ovarian failure. Welt, C.K., Hall, J.E., Adams, J.M., Taylor, A.E. J. Clin. Endocrinol. Metab. (2005) [Pubmed]
  9. Selective theca cell dysfunction in autoimmune oophoritis results in multifollicular development, decreased estradiol, and elevated inhibin B levels. Welt, C.K., Falorni, A., Taylor, A.E., Martin, K.A., Hall, J.E. J. Clin. Endocrinol. Metab. (2005) [Pubmed]
  10. Follicular arrest in polycystic ovary syndrome is associated with deficient inhibin A and B biosynthesis. Welt, C.K., Taylor, A.E., Fox, J., Messerlian, G.M., Adams, J.M., Schneyer, A.L. J. Clin. Endocrinol. Metab. (2005) [Pubmed]
  11. Evidence of early ovarian aging in fragile X premutation carriers. Welt, C.K., Smith, P.C., Taylor, A.E. J. Clin. Endocrinol. Metab. (2004) [Pubmed]
  12. Control of follicle-stimulating hormone by estradiol and the inhibins: critical role of estradiol at the hypothalamus during the luteal-follicular transition. Welt, C.K., Pagan, Y.L., Smith, P.C., Rado, K.B., Hall, J.E. J. Clin. Endocrinol. Metab. (2003) [Pubmed]
  13. Leptin and soluble leptin receptor in follicular fluid. Welt, C.K., Schneyer, A.L., Heist, K., Mantzoros, C.S. J. Assist. Reprod. Genet. (2003) [Pubmed]
  14. The physiology and pathophysiology of inhibin, activin and follistatin in female reproduction. Welt, C.K. Curr. Opin. Obstet. Gynecol. (2002) [Pubmed]
  15. Differential regulation of inhibin B and inhibin a by follicle-stimulating hormone and local growth factors in human granulosa cells from small antral follicles. Welt, C.K., Schneyer, A.L. J. Clin. Endocrinol. Metab. (2001) [Pubmed]
  16. Differential regulation of inhibin A and inhibin B by luteinizing hormone, follicle-stimulating hormone, and stage of follicle development. Welt, C.K., Smith, Z.A., Pauler, D.K., Hall, J.E. J. Clin. Endocrinol. Metab. (2001) [Pubmed]
 
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