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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)

D.L. Feldman

Novartis Institute for Biomedical Research

LSB 2195

Novartis Pharmaceuticals Corp.

556 Morris Ave.



Name/email consistency: high



  • Novartis Institute for Biomedical Research, LSB 2195, Novartis Pharmaceuticals Corp., 556 Morris Ave., USA. 2001
  • Metabolic and Cardiovascular Diseases, Pharmaceuticals Division, Novartis Institute for Biomedical Research, NJ 07901, USA. 1999 - 2000


  1. CGP 43371 paradoxically inhibits development of rabbit atherosclerotic lesions while inducing extra-arterial foam cell formation. Feldman, D.L., Sawyer, W.K., Jeune, M.R., Mogelesky, T.C., Von Linden-Reed, J., Forney Prescott, M. Atherosclerosis (2001) [Pubmed]
  2. Enhanced expression of renal endothelin-converting enzyme-1 and endothelin-A-receptor mRNA in rats with interstitial fibrosis following ureter ligation. Feldman, D.L., Mogelesky, T.C., Chou, M., Jeng, A.Y. J. Cardiovasc. Pharmacol. (2000) [Pubmed]
  3. Attenuation of puromycin aminonucleoside-induced glomerular lesions in rats by CGS 26303, a dual neutral endopeptidase/endothelin-converting enzyme inhibitor. Feldman, D.L., Mogelesky, T.C., Chou, M., Jeng, A.Y. J. Cardiovasc. Pharmacol. (2000) [Pubmed]
  4. The in vitro and ex vivo antioxidant properties, and hypolipidemic activity of CGP 2881. Feldman, D.L., Mogelesky, T.C., Sharif, R., Sawyer, W.K., Jeune, M., Hu, C.W., Leonards, K.S., Prescott, M.F. Atherosclerosis (1999) [Pubmed]
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