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David C. Warhurst

Pathogen Molecular Biology Unit

Department of Infectious and Tropical Diseases

London School of Hygiene and Tropical Medicine

University of London

[email]@lshtm.ac.uk

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Affiliations

Pathogen Molecular Biology Unit, Department of Infectious and Tropical Diseases, London School of Hygiene and Tropical Medicine, University of London, UK. 2003 - 2007
PHLS Malaria Laboratory, London School of Hygiene and Tropical Medicine, London WC1F 7HT. 2002

References

Activity of piperaquine and other 4-aminoquinoline antiplasmodial drugs against chloroquine-sensitive and resistant blood-stages of Plasmodium falciparum. Role of beta-haematin inhibition and drug concentration in vacuolar water- and lipid-phases. Warhurst, D.C., Craig, J.C., Adagu, I.S., Guy, R.K., Madrid, P.B., Fivelman, Q.L. Biochem. Pharmacol. (2007) [Pubmed]
Hydroxychloroquine is much less active than chloroquine against chloroquine-resistant Plasmodium falciparum, in agreement with its physicochemical properties. Warhurst, D.C., Steele, J.C., Adagu, I.S., Craig, J.C., Cullander, C. J. Antimicrob. Chemother. (2003) [Pubmed]
The relationship of physico-chemical properties and structure to the differential antiplasmodial activity of the cinchona alkaloids. Warhurst, D.C., Craig, J.C., Adagu, I.S., Meyer, D.J., Lee, S.Y. Malar. J. (2003) [Pubmed]
Polymorphism in the Plasmodium falciparum chloroquine-resistance transporter protein links verapamil enhancement of chloroquine sensitivity with the clinical efficacy of amodiaquine. Warhurst, D.C. Malar. J. (2003) [Pubmed]
Resistance to antifolates in Plasmodium falciparum, the causative agent of tropical malaria. Warhurst, D.C. Sci. Prog (2002) [Pubmed]