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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)

Deborah L. White

Haematology Department

Centre for Cancer Biology

SA Pathology (IMVS Campus)

University of Adelaide



Name/email consistency: high



  • Haematology Department, Centre for Cancer Biology, SA Pathology (IMVS Campus), University of Adelaide, Australia. 2010
  • Division of Haematology, SA Pathology IMVS/RAH Campus, Frome Road, Adelaide, Australia. 2009
  • Division of Hematology, Institute of Medical and Veterinary Science, Adelaide, Australia. 2005 - 2007


  1. Functional activity of the OCT-1 protein is predictive of long-term outcome in patients with chronic-phase chronic myeloid leukemia treated with imatinib. White, D.L., Dang, P., Engler, J., Frede, A., Zrim, S., Osborn, M., Saunders, V.A., Manley, P.W., Hughes, T.P. J. Clin. Oncol. (2010) [Pubmed]
  2. Predicting the response of CML patients to tyrosine kinase inhibitor therapy. White, D.L., Hughes, T.P. Curr. Hematol. Malig. Rep (2009) [Pubmed]
  3. Measurement of in vivo BCR-ABL kinase inhibition to monitor imatinib-induced target blockade and predict response in chronic myeloid leukemia. White, D., Saunders, V., Grigg, A., Arthur, C., Filshie, R., Leahy, M.F., Lynch, K., To, L.B., Hughes, T. J. Clin. Oncol. (2007) [Pubmed]
  4. Most CML patients who have a suboptimal response to imatinib have low OCT-1 activity: higher doses of imatinib may overcome the negative impact of low OCT-1 activity. White, D.L., Saunders, V.A., Dang, P., Engler, J., Venables, A., Zrim, S., Zannettino, A., Lynch, K., Manley, P.W., Hughes, T. Blood (2007) [Pubmed]
  5. OCT-1-mediated influx is a key determinant of the intracellular uptake of imatinib but not nilotinib (AMN107): reduced OCT-1 activity is the cause of low in vitro sensitivity to imatinib. White, D.L., Saunders, V.A., Dang, P., Engler, J., Zannettino, A.C., Cambareri, A.C., Quinn, S.R., Manley, P.W., Hughes, T.P. Blood (2006) [Pubmed]
  6. In vitro sensitivity to imatinib-induced inhibition of ABL kinase activity is predictive of molecular response in patients with de novo CML. White, D., Saunders, V., Lyons, A.B., Branford, S., Grigg, A., To, L.B., Hughes, T. Blood (2005) [Pubmed]
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