Christophe Depre
Cardiovascular Research Institute
Department of Cell Biology and Molecular Medicine
University of Medicine and Dentistry of New Jersey
New Jersey Medical School
Name/email consistency: high
- H11 Kinase/Heat Shock Protein 22 Deletion Impairs Both Nuclear and Mitochondrial Functions of STAT3 and Accelerates the Transition Into Heart Failure on Cardiac Overload. Qiu, H., Lizano, P., Laure, L., Sui, X., Rashed, E., Park, J.Y., Hong, C., Gao, S., Holle, E., Morin, D., Dhar, S.K., Wagner, T., Berdeaux, A., Tian, B., Vatner, S.F., Depre, C. Circulation (2011)
- H11 kinase prevents myocardial infarction by preemptive preconditioning of the heart. Depre, C., Wang, L., Sui, X., Qiu, H., Hong, C., Hedhli, N., Ginion, A., Shah, A., Pelat, M., Bertrand, L., Wagner, T., Gaussin, V., Vatner, S.F. Circ. Res. (2006)
- Characterization of pDJA1, a cardiac-specific chaperone found by genomic profiling of the post-ischemic swine heart. Depre, C., Wang, L., Tomlinson, J.E., Gaussin, V., Abdellatif, M., Topper, J.N., Vatner, S.F. Cardiovasc. Res. (2003)
- H11 kinase is a novel mediator of myocardial hypertrophy in vivo. Depre, C., Hase, M., Gaussin, V., Zajac, A., Wang, L., Hittinger, L., Ghaleh, B., Yu, X., Kudej, R.K., Wagner, T., Sadoshima, J., Vatner, S.F. Circ. Res. (2002)
