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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

Gregory A. Dissen

Division of Neuroscience

Oregon National Primate Research Center

505 North West 185th Avenue

Beaverton

USA

[email]@ohsu.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliation

  • Division of Neuroscience, Oregon National Primate Research Center, 505 North West 185th Avenue, Beaverton, USA. 2000 - 2012

References

  1. Hypothalamic EAP1 (enhanced at puberty 1) is required for menstrual cyclicity in nonhuman primates. Dissen, G.A., Lomniczi, A., Heger, S., Neff, T.L., Ojeda, S.R. Endocrinology (2012) [Pubmed]
  2. Excessive ovarian production of nerve growth factor facilitates development of cystic ovarian morphology in mice and is a feature of polycystic ovarian syndrome in humans. Dissen, G.A., Garcia-Rudaz, C., Paredes, A., Mayer, C., Mayerhofer, A., Ojeda, S.R. Endocrinology (2009) [Pubmed]
  3. Role of neurotrophic factors in early ovarian development. Dissen, G.A., Garcia-Rudaz, C., Ojeda, S.R. Semin. Reprod. Med. (2009) [Pubmed]
  4. In vivo manipulation of gene expression in non-human primates using lentiviral vectors as delivery vehicles. Dissen, G.A., Lomniczi, A., Neff, T.L., Hobbs, T.R., Kohama, S.G., Kroenke, C.D., Galimi, F., Ojeda, S.R. Methods (2009) [Pubmed]
  5. Expression of the insulin receptor-related receptor is induced by the preovulatory surge of luteinizing hormone in thecal-interstitial cells of the rat ovary. Dissen, G.A., Garcia-Rudaz, C., Tapia, V., Parada, L.F., Hsu, S.Y., Ojeda, S.R. Endocrinology (2006) [Pubmed]
  6. Neurotrophic control of ovarian development. Dissen, G.A., Romero, C., Paredes, A., Ojeda, S.R. Microsc. Res. Tech. (2002) [Pubmed]
  7. Intraovarian excess of nerve growth factor increases androgen secretion and disrupts estrous cyclicity in the rat. Dissen, G.A., Lara, H.E., Leyton, V., Paredes, A., Hill, D.F., Costa, M.E., Martinez-Serrano, A., Ojeda, S.R. Endocrinology (2000) [Pubmed]
  8. Direct effects of nerve growth factor on thecal cells from antral ovarian follicles. Dissen, G.A., Parrott, J.A., Skinner, M.K., Hill, D.F., Costa, M.E., Ojeda, S.R. Endocrinology (2000) [Pubmed]
 
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