Alessia Fornoni
Diabetes Research Institute
University of Miami L. Miller School of Medicine
Miami
Florida
USA
Name/email consistency: high
- Nephrin is expressed on the surface of insulin vesicles and facilitates glucose-stimulated insulin release. Fornoni, A., Jeon, J., Varona Santos, J., Cobianchi, L., Jauregui, A., Inverardi, L., Mandic, S.A., Bark, C., Johnson, K., McNamara, G., Pileggi, A., Molano, R.D., Reiser, J., Tryggvason, K., Kerjaschki, D., Berggren, P.O., Mundel, P., Ricordi, C. Diabetes (2010)
- Reversible bone pain and symmetric bone scan uptake in a dialysis patient treated with cinacalcet: a case report. Lenz, O., Sancassani, R., Bottino, C., Fornoni, A. J. Med. Case. Reports (2010)
- Inhibition of c-jun N terminal kinase (JNK) improves functional beta cell mass in human islets and leads to AKT and glycogen synthase kinase-3 (GSK-3) phosphorylation. Fornoni, A., Pileggi, A., Molano, R.D., Sanabria, N.Y., Tejada, T., Gonzalez-Quintana, J., Ichii, H., Inverardi, L., Ricordi, C., Pastori, R.L. Diabetologia (2008)
- The l-isoform but not d-isoforms of a JNK inhibitory peptide protects pancreatic beta-cells. Fornoni, A., Cobianchi, L., Sanabria, N.Y., Pileggi, A., Molano, R.D., Ichii, H., Rosero, S., Inverardi, L., Ricordi, C., Pastori, R.L. Biochem. Biophys. Res. Commun. (2007)
- Low insulin-like growth factor binding protein-2 expression is responsible for increased insulin receptor substrate-1 phosphorylation in mesangial cells from mice susceptible to glomerulosclerosis. Fornoni, A., Rosenzweig, S.A., Lenz, O., Rivera, A., Striker, G.E., Elliot, S.J. Endocrinology (2006)