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D. Jankovic

Immunobiology Section

Laboratory of Parasitic Diseases

National Institute of Allergy and Infectious Diseases

National Institutes of Health

USA

[email]@*.niaid.nih.gov

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Affiliation

Immunobiology Section, Laboratory of Parasitic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, USA. 1998 - 1999

References

Schistosome-infected IL-4 receptor knockout (KO) mice, in contrast to IL-4 KO mice, fail to develop granulomatous pathology while maintaining the same lymphokine expression profile. Jankovic, D., Kullberg, M.C., Noben-Trauth, N., Caspar, P., Ward, J.M., Cheever, A.W., Paul, W.E., Sher, A. J. Immunol. (1999) [Pubmed]
Optimal vaccination against Schistosoma mansoni requires the induction of both B cell- and IFN-gamma-dependent effector mechanisms. Jankovic, D., Wynn, T.A., Kullberg, M.C., Hieny, S., Caspar, P., James, S., Cheever, A.W., Sher, A. J. Immunol. (1999) [Pubmed]
CD4+ T cell-mediated granulomatous pathology in schistosomiasis is downregulated by a B cell-dependent mechanism requiring Fc receptor signaling. Jankovic, D., Cheever, A.W., Kullberg, M.C., Wynn, T.A., Yap, G., Caspar, P., Lewis, F.A., Clynes, R., Ravetch, J.V., Sher, A. J. Exp. Med. (1998) [Pubmed]