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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)

D. Jankovic

Immunobiology Section

Laboratory of Parasitic Diseases

National Institute of Allergy and Infectious Diseases

National Institutes of Health



Name/email consistency: high



  • Immunobiology Section, Laboratory of Parasitic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, USA. 1998 - 1999


  1. Schistosome-infected IL-4 receptor knockout (KO) mice, in contrast to IL-4 KO mice, fail to develop granulomatous pathology while maintaining the same lymphokine expression profile. Jankovic, D., Kullberg, M.C., Noben-Trauth, N., Caspar, P., Ward, J.M., Cheever, A.W., Paul, W.E., Sher, A. J. Immunol. (1999) [Pubmed]
  2. Optimal vaccination against Schistosoma mansoni requires the induction of both B cell- and IFN-gamma-dependent effector mechanisms. Jankovic, D., Wynn, T.A., Kullberg, M.C., Hieny, S., Caspar, P., James, S., Cheever, A.W., Sher, A. J. Immunol. (1999) [Pubmed]
  3. CD4+ T cell-mediated granulomatous pathology in schistosomiasis is downregulated by a B cell-dependent mechanism requiring Fc receptor signaling. Jankovic, D., Cheever, A.W., Kullberg, M.C., Wynn, T.A., Yap, G., Caspar, P., Lewis, F.A., Clynes, R., Ravetch, J.V., Sher, A. J. Exp. Med. (1998) [Pubmed]
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